Hyperactivity

# Overview
Hyperactivity can be described as a physical state in which a person is abnormally and easily excitable or exuberant. Strong emotional reactions, impulsive behaivor, and sometimes a short span of attention are also typical for a hyperactive person. Some individuals may show these characteristics naturally, as personality differs from person to person. Nonetheless, when hyperactivity starts to become a problem for the person or others, it may be classified as a medical disorder. The slang term  "hyper" is used to describe someone who is in a hyperactive state.

# Other causes of hyperactivity
There was a great deal of focus on Attention-deficit hyperactivity disorder as a cause of hyperactivity.  Other conditions can cause it as well.  Normal young children can be very lively and may or may not have short attention spans.  Normal teenagers can also appear hyperactive; puberty can cause it.  Children who are bored, are suffering from mental conflict, or are having problems at home - which may even include sexual abuse - can be hyperactive.  The disorder has a large range of effects on children.  Some have learning disabilities, while others may be very gifted, or both.
Hyperactivity can also occur because of problems with hearing or vision.  Overactive thyroid, lead poisoning, atypical depression, mania, anxiety, sleep deprivation and a range of psychiatric illnesses are some of the potential causes.
Severe cases of hyperactivity can be very harmful if left untreated, since hyperactive people seldom think about the consequences of their actions.

# Does sugar make one hyperactive?
A common belief is that eating too much sugar will make a person hyperactive. This belief is especially prevalent amongst parents and teachers who claim that children's behavior often get more rowdy, excited and energetic after they eat too many sugary food and drinks (such as candy or soda). One particular study found that the perception by parents regarding their children's hyperactivity depended on their belief as to whether they had been given sugar. Other studies have shown that the consumption of sugary items does not cause a measurable increase in hyperactive behavior. Hyperactivity is involved with attention span and personality traits.

# Other dietary causes of hyperactivity?
A September 2007 article from Southampton University touted by the British Food Standards Agency displayed that a statistically significant increase in the hyperactivity of children occurred after they consumed common artificial food colours and additives from fruit drinks. The list of compounds included the nearly ubiquitous additive in the beverage industry sodium benzoate and the also popular tartrazine, along with quinonline yellow, sunset yellow, carmoisine and allura red. The British Food Standard Agency has revised its stance on these additives; informing parents of children that demonstrate hyperactive behaviour that removal of foods contain the six additives from their diet could have beneficial results on behaviour.
Other studies have recommended the Feingold Diet which eliminates several synthetic colors, synthetic flavors, synthetic preservatives, and artificial sweeteners.  Scientific studies have shown mixed results in double blind studies of the diet, but recently several reports have been published indicating a statistically significant effect on the behaviour of children on the diet .

# Causes

## Common Causes
- ADHD
- Alcoholism
- Amphetamine
- Atomoxetine
- Bipolar disorder
- Brain tumor
- Cerebral palsy
- Drug Abuse
- Hyperthyroidism
- Stress
- Substance abuse

## Causes by Organ System

## Causes in Alphabetical Order
- 49,XXXXY syndrome
- Adrenoleukodystrophy
- Alcoholism
- Allergic irritability syndrome
- Allergic tension-fatigue syndrome
- Amoxicillin
- Amphetamine
- Anorexia Nervosa
- Arginase deficiency
- Armodafinil
- Atomoxetine
- Attention Deficit Hyperactivity Disorder
- Autism
- Bipolar
- Brain tumor
- Bupropion
- Cerebral palsy
- Chlorpromazine
- Chromosome 15 inverted duplication
- Chromosome 17, trisomy 17p11.2
- Chromosome 17p, partial deletion
- Chromosome 22 Ring
- Chromosome 8p deletion syndrome
- Chromosome 9, trisomy 9q
- Chromosome 9q duplication
- Citrullinemia
- Clonidine
- Dexedrine overdose
- Diencephalic syndrome
- Dubowitz syndrome
- English Ivy poisoning
- Ethosuximide
- Fetal alcohol syndrome
- Fluphenazine
- Focal epilepsy syndrome
- Fragile-X Syndrome
- Graves Disease
- Guanfacine
- Haloperidol
- High T4 syndrome
- Lead poisoning
- Lissencephaly
- Loratadine
- Mania
- Methylphenidate
- Modafinil
- Mucopolysaccharidosis
- Nabilone
- Nicardipine
- Occupational lead exposure
- Phenol sulfotransferase deficiency
- Phenylketonuria
- Pitt-Rogers-Danks syndrome
- Potocki-Lupski syndrome
- Reboxetine
- Seckel syndrome
- Smith-Magenis syndrome
- Strauss syndrome
- Stress
- Subacute Thyroiditis
- Thorazine
- Tolcapone
- Tuberous sclerosis