OpenLLM-France/wikimedia huggingface dataset:
# Dariyapur, Nalanda

Dariyapur is a village situated in the heart of Nalanda district in Bihar state, India. It is 9 km far from its local market Hilsa which is its subdivision also. Lohanda is the nearest railway halt from here.

## Introduction

Agriculture, commerce and services are the main employment sources in the village. The main crops are paddy, wheat, corn and some rabi crops.
The name of this village "dariyapur" is made of two words "dariya" & "pur" means village of rivers. There are three local rivers named: Dorwa, Jankariya and Sindh merge in the western side of this village. The village is situated in the Hilsa subdivision and the Kachhiyawan panchayat. The police station is in Nagarnausa, and the local post office is in Kachhiyawan.
Nearby villages include Barabigha, Bhobhi, kachhiyawan and Bhudkur. This village has a great history. And its countrymen are very kindhearted as well as industrious. Thus its versatile development is going on. Now, signs of development are visible such as government school, electricity, water supply, double-lane solid roads, and Internet availability.
More than 80 % people belong to Kurmi caste and engaged primarily in agriculture. In recent times, government employment and self-business has increased. Notably, this village has produced few scientists recognized globally.
# Hitts & Mrs.

Hitts & Mrs. (2004) is the second novel by Emmy-winning author Lori Bryant-Woolridge. It made the Essence Magazine bestseller list. Hitts & Mrs. built on the positive reviews and sales figures for Bryant-Woolridge's first book, Read Between the Lies.

## Edition

* HarperCollins Trade Paperback (January 2004), 367pp., ISBN 0-06-054059-1.
# CHSY1

Chondroitin sulfate synthase 1 is an enzyme that in humans is encoded by the CHSY1 gene.
CHSY1 synthesizes chondroitin sulfate, a glycosaminoglycan expressed on the surface of most cells and in extracellular matrices. Glycosaminoglycan chains are covalently linked to a wide range of core protein families and regulate many biologic processes, including cell proliferation and recognition, extracellular matrix deposition, and morphogenesis.
# Dedicator of cytokinesis protein 3

Dedicator of cytokinesis protein 3 (Dock3), also known as MOCA (modifier of cell adhesion) and PBP (presenilin-binding protein), is a large (~180 kDa) protein encoded in the human by the DOCK3 gene, involved in intracellular signalling networks. It is a member of the DOCK-B subfamily of the DOCK family of guanine nucleotide exchange factors (GEFs) which function as activators of small G-proteins. Dock3 specifically activates the small G protein Rac.

## Discovery

Dock3 was originally discovered in a screen for proteins that bind presenilin (a transmembrane protein which is mutated in early onset Alzheimer's disease). Dock3 is specifically expressed in neurons (primarily in the cerebral cortex and hippocampus).

## Structure and function

Dock3 is part of a large class of proteins (GEFs) which contribute to cellular signalling events by activating small G proteins. In their resting state G proteins are bound to Guanosine diphosphate (GDP) and their activation requires the dissociation of GDP and binding of guanosine triphosphate (GTP). GEFs activate G proteins by promoting this nucleotide exchange.
Dock3 exhibits the same domain arrangement as Dock180 (a member of the DOCK-A subfamily and the archetypal member of the DOCK family) and these proteins share a considerable (40%) degree of sequence similarity.

## Regulation

Since Dock3 shares the same domain arrangement as Dock180 it is predicted to have a similar array of binding partners, although this has yet to be demonstrated. It contains an N-terminal SH3 domain, which in Dock180 binds ELMO (a family of adaptor proteins which mediate recruitment and efficient GEF activity of Dock180), and a C-terminal proline-rich region which, in Dock180, binds the adaptor protein CRK.

## Downstream signalling

Dock3 GEF activity is directed specifically at Rac1. Dock3 has not been shown to interact with Rac3, another Rac protein which is expressed in neuronal cells, and this may be because Rac3 is primarily located in the perinuclear region. In fact, Rac1 and Rac3 appear to have distinct and antagonistic roles in these cells. Dock3-mediated Rac1 activation promotes reorganisation of the cytoskeleton in SH-SY5Y neuroblastoma cells and primary cortical neurones as well as morphological changes in fibroblasts. It has also been shown to regulate neurite outgrowth and cell-cell adhesion in B103 and PC12 cells.

## In neurological disorders

The first indication that Dock3 might be involved in neurological disorders came when Dock3 was shown to bind to presenilin, a transmembrane enzyme involved in the generation of beta amyloid (Aβ), accumulation of which is an important step in the development of Alzheimer's disease. Dock3 has been shown to undergo redistribution and association with neurofibrillary tangles in brain samples from patients with Alzheimer's disease. A mutation in Dock3 was also identified in a family displaying a phenotype resembling attention-deficit hyperactivity disorder (ADHD).
# Exonuclease 1

Exonuclease 1 is an enzyme that in humans is encoded by the EXO1 gene.
This gene encodes a protein with 5' to 3' exonuclease activity as well as RNase activity (endonuclease activity cleaving RNA on DNA/RNA hybrid). It is similar to the Saccharomyces cerevisiae protein Exo1 which interacts with Msh2 and which is involved in DNA mismatch repair and homologous recombination. Alternative splicing of this gene results in three transcript variants encoding two different isoforms.

## Meiosis

ExoI is essential for meiotic progression through metaphase I in the budding yeast Saccharomyces cerevisiae and in mouse.
Recombination during meiosis is often initiated by a DNA double-strand break (DSB) as illustrated in the accompanying diagram. During recombination, sections of DNA at the 5' ends of the break are cut away in a process called resection. In the strand invasion step that follows, an overhanging 3' end of the broken DNA molecule "invades" the DNA of a homologous chromosome that is not broken, forming a displacement loop (D-loop). After strand invasion, the further sequence of events may follow either of two main pathways leading to a crossover (CO) or a non-crossover (NCO) recombinant (see Genetic recombination and Homologous recombination). The pathway leading to a CO involves a double Holliday junction (DHJ) intermediate. Holliday junctions need to be resolved for CO recombination to be completed.
During meiosis in S. cerevisiae, transcription of the Exo1 gene is highly induced. In meiotic cells, Exo1 mutation reduces the processing of DSBs and the frequency of COs. Exo1 has two temporally and biochemically distinct functions in meiotic recombination. First, Exo1 acts as a 5’–3’ nuclease to resect DSB-ends. Later in the recombination process, Exo1 acts to facilitate the resolution of DHJs into COs, independently of its nuclease activities. In resolving DHJs, Exo 1 acts together with MLH1-MLH3 heterodimer (MutL gamma) and Sgs1 (ortholog of Bloom syndrome helicase) to define a joint molecule resolution pathway that produces the majority of crossovers.
Male mice deficient for Exo1 are capable of normal progress through the pachynema stage of meiosis, but most germ cells fail to progress normally to metaphase I due to dynamic loss of chiasmata. Surprisingly though, this meiotic role of Exo1 is not mediated by its nuclease activity per se, since Exo1-DA mice harboring a point mutation in Exo1's nuclease domain have no detectable meoitic defects.

## Interactions

Exonuclease 1 has been shown to interact with MSH2 and MLH1.
# HIST1H4H

Histone H4 is a protein that in humans is encoded by the HIST1H4H gene.
Histones are basic nuclear proteins that are responsible for the nucleosome structure of the chromosomal fiber in eukaryotes. Two molecules of each of the four core histones (H2A, H2B, H3, and H4) form an octamer, around which approximately 146 bp of DNA is wrapped in repeating units, called nucleosomes. The linker histone, H1, interacts with linker DNA between nucleosomes and functions in the compaction of chromatin into higher order structures. This gene is intronless and encodes a member of the histone H4 family. Transcripts from this gene lack polyA tails but instead contain a palindromic termination element. This gene is found in the large histone gene cluster on chromosome 6.
# Racine Legion

The Racine Legion was a professional American football team based in Racine, Wisconsin, of the National Football League from 1922 to 1924. Its official name was the Horlick-Racine Legion. The team then operated as the Racine Tornadoes in 1926.

## History

In 1915, the Racine Regulars formed Wisconsin's first important semi-professional team. They primarily played against teams from Illinois and Indiana. The team became known as the Racine Battery C in 1916 after many of the players joined the First Wisconsin Reserve Artillery Battery C.
Because of World War I and the Spanish flu pandemic of 1918 the team took a break. It was reorganized in 1919 with sponsorship from the local American Legion post and William Horlick, president of his family's malted milk company. The reorganized team was known as the Horlick-Racine Legion.
In 1922, the American Professional Football Association changed its name to the National Football League. Racine, now known simply as the Racine Legion, although its official name remained the Horlick-Racine Legion, was one of four new teams admitted to membership that season.
Led by the fullback-kicker Hank Gillo, who led the league in scoring with 52 points, Racine finished sixth in the 18-team league with a 6–4–1 record. Despite two more respectable seasons, the team failed because of finances. In 1925, the franchise was held over by the NFL, but they did not field a team that season.
Milton "Mitt" Romney, team captain at the University of Chicago in 1922, became a quarterback for the Racine Legion before later joining the Chicago Bears. He was a cousin of Michigan Governor George W. Romney, and his nickname inspired that of George Romney's son, 2012 U.S. presidential candidate Mitt Romney.
Facing the threat of Red Grange's American Football League in 1926, the NFL was eager to get as many teams and players as possible into the fold to keep them away from the AFL. The Racine franchise was reactivated. The team, now called the Tornadoes, had a few of the same players as the Legion but many stars went to other teams in 1925. After winning their first game, the Tornadoes lost four in a row and disbanded in late October because of struggling finances.

## Season-by-season

| | Year | W | L | T | Finish | Coach |
| Legion | 1922 | 6 | 4 | 1 | 6th | Babe Ruetz |
| Legion | 1923 | 4 | 4 | 2 | 9th | Babe Ruetz |
| Legion | 1924 | 4 | 3 | 3 | 7th | Babe Ruetz |
| Legion | 1925 | Suspended Operations | Suspended Operations | Suspended Operations | Suspended Operations | Suspended Operations |
| Tornadoes | 1926 | 1 | 4 | 0 | 16th | Shorty Barr, Wally McIlwain |

## Trivia

The Racine Legion have the distinction of winning the only NFL game where one team scored exactly 4 points. On November 25, 1923, the Legion beat the Chicago Cardinals by a score of 10–4. Further, the Cardinals franchise had used Racine as its geographic name until 1922, referring to Racine Ave. on which the team's home field was located.
# OR6C1

Olfactory receptor 6C1 is a protein that in humans is encoded by the OR6C1 gene.
Olfactory receptors interact with odorant molecules in the nose, to initiate a neuronal response that triggers the perception of a smell. The olfactory receptor proteins are members of a large family of G-protein-coupled receptors (GPCR) arising from single coding-exon genes. Olfactory receptors share a 7-transmembrane domain structure with many neurotransmitter and hormone receptors and are responsible for the recognition and G protein-mediated transduction of odorant signals. The olfactory receptor gene family is the largest in the genome. The nomenclature assigned to the olfactory receptor genes and proteins for this organism is independent of other organisms.
# THOC5

THO complex subunit 5 homolog is a protein that in humans is encoded by the THOC5 gene. THOCs is a member of THO complex which is a subcomplex of the transcription/export complex (TREX).
THOC5 is evolutionarily conserved in higher eukaryotes, however the exact roles of THOC5 in transcription and mRNA export are still unclear. THOC5 is phosphorylated by several protein kinases at multiple residues upon extracellular stimuli. These include stimulation with growth factors/cytokines/chemokines, or DNA damage reagents. Furthermore, THOC5 is a substrate for several oncogenic tyrosine kinases, suggesting that THOC5 may be involved in cancer development.
Recent THOC5 knockout mouse data reveal that THOC5 is an essential element in the maintenance of stem cells and growth factor/cytokine-mediated differentiation/proliferation. Furthermore, depletion of THOC5 influences less than 1% of total mRNA export in the steady state, however it influences more than 90% of growth factor/cytokine induced genes. THOC5, thereby contributes to the 3′ processing and/or export of immediate-early genes induced by extracellular stimuli. These studies bring new insight into the link between the mRNA export complex and immediate-early gene response. The data from these studies also suggest that THOC5 may be a useful tool for studying stem cell biology, for modifying the differentiation processes and for cancer therapy.
# William Smillie

William Smillie (c. 1810 – 11 Dec 1852) was an appointed member of the first Legislative Council of South Australia, serving from March 1840 to February 1851.

## History

Smillie was a son of Matthew Smillie (c. 1781 – 12 March 1847), a solicitor of Leith, Scotland and his wife Elizabeth Corse Smillie, née Nairne (c. 1791 – 17 October 1861). Smillie was a partner in his father's legal practice. He became fascinated with the newly founded colony of South Australia, and published a series of articles in the Stirling Advertiser, reprinted as a book The Great South Land in 1838.
He and his parents subsequently emigrated on the Indus, arriving in South Australia in January 1839. His father purchased three sections in the vicinity of Mount Barker, which he subdivided, establishing the township of Nairne which he named in honour of his wife. They established a home, "The Vallies" or "The Valleys" in the area. In 1840 Smillie was appointed advocate-general, which position until 1857 was linked to a seat on the Legislative Council, succeeding Robert Bernard.
Smillie was plagued with poor health. In 1849 he took three months' leave of absence in Tasmania, in the hope of some improvement. William Bartley acted in his absence. His health further failing, in June 1851 Smillie took eighteen months' leave of absence, holidaying in St. Thomas in the West Indies, where he experienced some improvement. Charles Mann (the first incumbent) acted in his absence for a month or two, followed by Richard Davies Hanson. He died in Paris, en route to Italy, and was buried in Scotland.
   He was distinguished by his general amiableness and cultivated mind, and by the interest he took, not only in his professional duties, but in all that related to the well-being of the colony

## Family

William Smillie married Eliza Jane Farquharson ( – ) daughter of Robert Farquarson of Allargue, in 1844. their family included:
* Jean Nairne Smillie (16 April 1846 – 29 February 1880)
* Elizabeth Smillie (c. 1848 – 4 September 1869)
* Matthew William Smillie (23 February 1850 – 23 October 1867) died in Aberdeen
# VASH1

Vasohibin-1 is a protein that in humans is encoded by the VASH1 gene.
# Acid alpha-glucosidase

Acid alpha-glucosidase, also called acid maltase, is an enzyme that helps to break down glycogen in the lysosome. It is functionally similar to glycogen debranching enzyme, but is on a different chromosome, processed differently by the cell and is located in the lysosome rather than the cytosol. In humans, it is encoded by the GAA gene. Errors in this gene cause glycogen storage disease type II (Pompe disease).

## Function

This gene encodes lysosomal alpha-glucosidase, which is essential for the degradation of glycogen to glucose in lysosomes. Different forms of acid alpha-glucosidase are obtained by proteolytic processing. Defects in this gene are the cause of glycogen storage disease II, also known as Pompe disease, which is an autosomal recessive disorder with a broad clinical spectrum. Three transcript variants encoding the same protein have been found for this gene.
# Brain-specific angiogenesis inhibitor 1

Brain-specific angiogenesis inhibitor 1 is a protein that in humans is encoded by the BAI1 gene. It is a member of the adhesion-GPCR family of receptors.

## Function

Angiogenesis is controlled by a local balance between stimulators and inhibitors of new vessel growth and is suppressed under normal physiologic conditions. Angiogenesis has been shown to be essential for growth and metastasis of solid tumors. In order to obtain blood supply for their growth, tumor cells are potently angiogenic and attract new vessels as results of increased secretion of inducers and decreased production of endogenous negative regulators. BAI1 contains at least one 'functional' p53-binding site within an intron, and its expression has been shown to be induced by wildtype p53. There are two other brain-specific angiogenesis inhibitor genes, designated BAI2 and BAI3 which along with BAI1 have similar tissue specificities and structures, however only BAI1 is transcriptionally regulated by p53. BAI1 is postulated to be a member of the secretin receptor family, an inhibitor of angiogenesis and a growth suppressor of glioblastomas.

## Interactions

Brain-specific angiogenesis inhibitor 1 has been shown to interact with BAIAP3 and MAGI1.
# Thou Shalt Not Kill (TV series)

Thou Shalt Not Kill (Italian: Non uccidere) is an Italian crime drama television series starring Miriam Leone. The first season aired in Italy on Rai 3 in 2017, while the second season premiered on Rai's streaming service RaiPlay. Each season consists of 12 episodes. In the U.S. and U.K., the series is available on Walter Presents. This series aired on American PBS stations in fall 2019, and again in 2021.

## Synopsis

Thou Shalt Not Kill is an Italian detective drama set in Turin. The series follows Valeria Ferro, who investigates murders with the help of her colleagues in the police department, while also uncovering secrets from her own family's past. Valeria's mother was imprisoned for killing her husband when Valeria was a child.
The show also explores Valeria's complex relationships with her family and colleagues, which often intersect, especially since her mother was convicted by the same police department in which Valeria works.

## Cast

* Miriam Leone as Valeria Ferro
* Monica Guerritore as Lucia Ferro
* Matteo Martari as Andrea Russo
* Thomas Trabacchi as Giorgio Lombardi
* Riccardo Lombardo as Gerardo Mattei
* Luca Terracciano as Luca Rinaldi
* Davide Iacopini as Giacomo Ferro
* Viola Sartoretto as Michela Ferro
* Gigio Alberti as Giulio Ferro
* Crystal De Glaudi as Costanza Ferro

## Broadcast

This was first broadcast on Rai 3 in Italy for seasons one and two, with each season containing 12 episodes, starting in 2017. Season two premiered on Rai's streaming service RaiPlay. In the U.S. and U.K., the series is available on Walter Presents. The show debuted on American PBS stations in the fall of 2019, airing in Italian with English subtitles.
# Halaku (film)

Halaku is a 1956 Hindi language film with Pran in the title role playing Hulagu.
The cast includes Meena Kumari, Ajit, Minoo Mumtaz, Raj Mehra, Shammi and Veena.

## Plot

Halaku (Pran), the powerful emperor of Iran, rules the country wisely and with an iron hand. When he comes upon one of his subjects, Niloufer (Meena Kumari), he stakes his claim on her and wants her to be his wife, despite his wife (Veena) opposing his marriage plans. Niloufer, who loves Pervez (Ajit Khan), refuses to submit to Halaku. He turns his wrath on both Niloufer, Pervez, and his wife. Will Niloufer and Pervez survive the ruthless Halaku?

## Music

* "Use Mil Gayi Nayi Zindagi" - Lata Mangeshkar
* "Aaja Ke Intizar Me, Jane Ko Hai Bahar Bhi" - Lata Mangeshkar, Mohammed Rafi
* "Yeh Chand Yeh Sitare Yeh Sath Teraa Mera" - Lata Mangeshkar
* "Aji Chale Aao, Tumhe Aankho Ne Dil Me Bulaya" - Lata Mangeshkar, Asha Bhosle
* "Bol Mere Malik Teraa Kya Yahee Hai Insaf" - Lata Mangeshkar
* "O Sunta Ja, Dil Ka Nagma Pyar Ki Dhun Par" - Lata Mangeshkar
* "Dil Ka Na Karna Aitbar Koi" - Mohammed Rafi, Lata Mangeshkar
* "Teri Duniya Se Jate Hain, Chupaye Dil Me Gam Apna" - Lata Mangeshkar

## Cast

* Pran as Sultan Halaku Khan
* Ajit as Parvez
* Meena Kumari as Nilofar Nadir
* Minoo Mumtaz
* Raj Mehra
* Sunder
* Shammi: Navbahar
* Veena: Maharani
* Niranjan Sharma
* Helen as Dancer
# 1995 WDC World Darts Championship

The 1995 Proton Cars World Darts Championship was the second world championship organised by the World Darts Council (now the Professional Darts Corporation). It was held between 27 December 1994 and 2 January 1995.
Phil Taylor emerged as champion, overcoming John Lowe 5–4 in the semi-final before beating Rod Harrington 6–2 in the final to claim his first WDC World Championship title, and his third World Championship in all.

## Seeds

There were eight seeds in the World Championship.
* Dennis Priestley
* Rod Harrington
* Peter Evison
* Phil Taylor
* Bob Anderson
* Kevin Spiolek
* Jamie Harvey
* Alan Warriner

## Prize money

| Position (num. of players) | Position (num. of players) | Prize money (Total: £55,000) |
| Winner | (1) | £12,000 |
| Runner-Up | (1) | £6,000 |
| Third place | (1) | £4,000 |
| Fourth place | (1) | £3,000 |
| Quarter-finalists | (4) | £2,000 |
| Second place in group | (8) | £1,500 |
| Third place in group | (8) | £1,250 |

## Results
### Group stage

Players in bold denote match winners.

### Knockout stages

Players in bold denote match winners.
| Quarter-finals (best of 7 sets) 29–30 December | Quarter-finals (best of 7 sets) 29–30 December | Quarter-finals (best of 7 sets) 29–30 December | Semi-finals (best of 9 sets) 1 January | Semi-finals (best of 9 sets) 1 January | Semi-finals (best of 9 sets) 1 January | Final (best of 11 sets) 2 January | Final (best of 11 sets) 2 January | Final (best of 11 sets) 2 January |
| A1 | Peter Evison (3) 88.61 | 4 | | | | | | |
| B1 | Kevin Spiolek (6) 82.85 | 1 | | | | | | |
| B1 | Kevin Spiolek (6) 82.85 | 1 | A1 | Peter Evison (3) 82.43 | 1 | | | |
| | | | D1 | Rod Harrington (2) 82.29 | 5 | | | |
| C1 | Jamie Harvey (7) 87.42 | 2 | D1 | Rod Harrington (2) 82.29 | 5 | | | |
| D1 | Rod Harrington (2) 91.40 | 4 | | | | | | |
| D1 | Rod Harrington (2) 91.40 | 4 | | | | D1 | Rod Harrington (2) 87.15 | 2 |
| | | | | | | F1 | Phil Taylor (4) 94.12 | 6 |
| E1 | Bob Anderson (5) 84.44 | 1 | | | | | | | F1 | Phil Taylor (4) 94.12 | 6 |
| F1 | Phil Taylor (4) 89.70 | 4 | | | |
| F1 | Phil Taylor (4) 89.70 | 4 | F1 | Phil Taylor (4) 96.22 | 5 | Third place play-off (best of 7 sets) | Third place play-off (best of 7 sets) | Third place play-off (best of 7 sets) |
| | | | H1 | John Lowe 90.86 | 4 |
| G1 | Dennis Smith 89.23 | 0 | H1 | John Lowe 90.86 | 4 | A1 | Peter Evison (3) 84.95 | 2 |
| H1 | John Lowe 96.77 | 4 | | | | H1 | John Lowe 82.81 | 4 |