Miracle-dz/UniFAR-FTUs huggingface dataset:
Teams for Teams Performance in Multi-Human/Multi-Robot Teams

The present study addresses the interaction between automation and organization of human teams in controlling large robot teams performing an Urban Search and Rescue (USAR) task. We identify three subtasks: perceptual - visual search for victims, assistance - teleoperation to assist robot, and navigation - path planning and coordination. For the studies reported, navigation was selected for automation because it involves weak dependencies among robots making it more complex and because it was shown in an earlier experiment to be the most difficult. Two possible ways to organize operators were identified as assignment of robots to particular operators or as a shared pool in which operators service robots from the population as needed. The experiment compares two member teams of operators controlling teams of 12 robots each, in the assigned robots conditions or sharing control of 24 robots in the shared pool conditions using either waypoint control or autonomous path planning. Automating path planning improved system performance. Effects of team organization were equivocal.
Social Consensus Feedback as a Strategy to Overcome Spontaneous Gender Stereotypes

Across two experiments the present research examined the use of social consensus feedback as a strategy for overcoming spontaneous gender stereotyping when certain social role nouns and professional terms are read. Participants were presented with word pairs comprising a role noun (e.g., surgeon) and a kinship term (e.g., mother) and asked to decide whether both terms could refer to the same person. In the absence of training, participants responded more slowly and less accurately to stereotype incongruent pairings (e.g., surgeon/mother) than stereotype congruent pairings (e.g., surgeon/father). When participants were provided with (fictitious) social consensus feedback, constructed to suggest that past participants did not succumb to stereotypes, performance to incongruent pairings improved significantly (Experiment 1). The mechanism(s) through which the social feedback operated were then investigated (Experiment 2), with results suggesting that success was due to social compliance processes. Implications of findings for the field of discourse processing are discussed.
Indoor localization methods based on Wi-Fi lateration and signal strength data collection

The paper describes two Wi-Fi lateration methods based on signal propagation model and signal strength data collection for indoor localization using Android-based mobile device. The considered methods use log-normal path loss model for signal propagation and received signal strength measurement collection for distance estimation and lateration approach for localization. The indoor signal propagation problem is resolved by received signal strength measuring and special ring radio map building those improve localization accuracy. Indoor localization technique opens possibilities for development various intelligent systems that provide the user location-based information inside buildings.
Sleep disturbance as part of the neurofibromatosis type 1 phenotype in adults

Neurofibromatosis type 1 (NF1) is an autosomal dominant condition with a wide array of neurological complications, including cognitive dysfunction, tumors, malformations, neuropathy, neurovascular disease, and epilepsy. Many of these complications may impact on sleep quality and cause sleep disturbance. Previously sleep disturbance in NF1 has been specifically addressed solely in children. We performed a prospective study of sleep quality in 114 consecutive out‐patients with NF1 attending our national neurofibromatosis service. The Epworth sleepiness scale (ESS) and the Pittsburgh sleep quality index (PSQI) were administered, and information was obtained from patient records on drugs potentially impacting on sleep, complications directly affecting sleep and employment status. The mean ESS was 6.8, and 21% had an abnormally high ESS of 10 or more. The mean global PSQI score was 8.4 (norm mean 2.67), with abnormally high scores in all sleep domains. Thirty‐nine patients had a bed partner and 54% reported features suggestive of periodic limb movements of sleep, 43% had features suggestive of obstructive sleep apnoea, and 10.8% experienced confusion on waking. There was no evidence of phase shift. The ESS did not correlate with the PSQI, but unemployment status was associated with worse global PSQI score and multiple domain sub‐scales of sleep quality in the PSQI. We conclude that sleep disturbance and poor sleep quality are significantly more frequent in the adult NF1 patient population. It is likely to be multi‐factorial, related to pain, anxiety, depression, cognitive issues, and organic sleep pathology. We recommend careful assessment of patients to determine underlying triggers and possible treatment strategies. © 2013 Wiley Periodicals, Inc.
Impaired healing of gastric lesions in streptozotocin-induced diabetic rats: effect of basic fibroblast growth factor.

We examined the influence of diabetes on the healing of HCI-induced gastric lesions and the healing promoting effect of basic fibroblast growth factor (bFGF) on these lesions under diabetic conditions induced in rats by streptozotocin (70 mg/kg, i.p.). The experiments were performed using 2-wk streptozocin-diabetic rats with blood glucose levels of >300 mg/dl. After 18 hr fasting, these animals were given 1 ml of 0.6 N HCl by gavage, and 1 hr later they were fed normally before being killed on various days after HCI treatment. Recombinant human basic fibroblast growth factor (acid resistant recombinant human basic fibroblast growth factor mutein CS-23: 10 to 1000 ng/kg x 2, p.o.) or insulin (4 U/rat x 1, s.c.) was given 5 days after HCl treatment. Gastric lesions induced by HCI healed to quiescent state within 5 days both macro- and microscopically. Diabetic conditions did not affect the development of HCI-induced gastric lesions but significantly delayed the healing of these lesions. Daily administration of insulin returned high blood glucose levels to within normal ranges (120-140 mg/dl) and significantly antagonized the delayed healing of these lesions. The delayed healing in diabetic rats was also significantly promoted by recombinant human basic fibroblast growth factor (>300 ng/kg x 2) without any effect on blood glucose level. In normal rats, the mucosal levels of bFGF increased significantly in response to gastric injury at 3 days after HCI treatment. The mucosal bFGF levels in streptozotocin-diabetic rats were significantly lower under basal conditions before HCI treatment and did not increase after injury, yet such dysregulation of bFGF production was partially restored by insulin treatment. rhbFGF even at 1000 ng/kg had no effect on gastric acid secretion in either normal or streptozotocin-diabetic rats. These results suggest that diabetic conditions have deleterious influences on the healing of acute gastric lesions in both an insulin- and bFGF-sensitive manner, and that the administration of exogenous bFGF antagonizes the delayed healing of gastric lesions observed in diabetic animals.
Amantadine as treatment for dyskinesias and motor fluctuations in Parkinson's disease

Objective: To determine the effects of the N-methyl-D-aspartate (NMDA) antagonist amantadine on levodopa-associated dyskinesias and motor fluctuations in Parkinson's disease (PD). Background: NMDA receptor blockade can ameliorate levodopa-induced dyskinesias in primates and PD patients. Amantadine, a well-tolerated and modestly effective antiparkinsonian agent, was recently found to possess NMDA antagonistic properties. Methods: Eighteen patients with advanced PD participated in a double-blind, placebo-controlled, cross-over study. At the end of each 3-week treatment arm, parkinsonian and dyskinesia scores were obtained during a steady-state intravenous levodopa infusion. Motor fluctuations and dyskinesias were also documented with patient-kept diaries and Unified Parkinson's Disease Rating Scale (UPDRS) interviews. Results: In the 14 patients completing this trial, amantadine reduced dyskinesia severity by 60% (p = 0.001) compared to placebo, without altering the antiparkinsonian effect of levodopa. Motor fluctuations occurring with patients' regular oral levodopa regimen also improved according to UPDRS and patient-kept diaries. Conclusions: These findings suggest that amantadine given as adjuvant to levodopa can markedly improve motor response complications and support the view that hyperfunction of NMDA receptors contributes to the pathogenesis of levodopa-associated motor complications.
One-sided underreaction in stock prices

In this paper, we will add new evidence for the proposition that the Efficient Market Hypothesis is violated in financial markets. Nearly all stocks in the Euro Stoxx 50 have the characteristic that if they perform one day extremely bad, they will tend to perform the following day also extremely bad. Moreover, this underreaction effect does not occur when a stock performs exceptionally well on a certain day. The usual arguments put forward to argue that market efficiency holds notwithstanding this phenomenon are discussed, together with the existing models in the literature which produce under and overreaction. It will turn out that these models lack explanatory power for our results. For this reason, we propose a design for a model that probably could explain one-sided underreaction.
Perilipin regulates the thermogenic actions of norepinephrine in brown adipose tissue Published, JLR Papers in Press, March 30, 2007.

In response to cold, norepinephrine (NE)-induced triacylglycerol hydrolysis (lipolysis) in adipocytes of brown adipose tissue (BAT) provides fatty acid substrates to mitochondria for heat generation (adaptive thermogenesis). NE-induced lipolysis is mediated by protein kinase A (PKA)-dependent phosphorylation of perilipin, a lipid droplet-associated protein that is the major regulator of lipolysis. We investigated the role of perilipin PKA phosphorylation in BAT NE-stimulated thermogenesis using a novel mouse model in which a mutant form of perilipin, lacking all six PKA phosphorylation sites, is expressed in adipocytes of perilipin knockout (Peri KO) mice. Here, we show that despite a normal mitochondrial respiratory capacity, NE-induced lipolysis is abrogated in the interscapular brown adipose tissue (IBAT) of these mice. This lipolytic constraint is accompanied by a dramatic blunting (∼70%) of the in vivo thermal response to NE. Thus, in the presence of perilipin, PKA-mediated perilipin phosphorylation is essential for NE-dependent lipolysis and full adaptive thermogenesis in BAT. In IBAT of Peri KO mice, increased basal lipolysis attributable to the absence of perilipin is sufficient to support a rapid NE-stimulated temperature increase (∼3.0°C) comparable to that in wild-type mice. This observation suggests that one or more NE-dependent mechanism downstream of perilipin phosphorylation is required to initiate and/or sustain the IBAT thermal response.
Developing crossmodal expression recognition based on a deep neural model

A robot capable of understanding emotion expressions can increase its own capability of solving problems by using emotion expressions as part of its own decision-making, in a similar way to humans. Evidence shows that the perception of human interaction starts with an innate perception mechanism, where the interaction between different entities is perceived and categorized into two very clear directions: positive or negative. While the person is developing during childhood, the perception evolves and is shaped based on the observation of human interaction, creating the capability to learn different categories of expressions. In the context of human–robot interaction, we propose a model that simulates the innate perception of audio–visual emotion expressions with deep neural networks, that learns new expressions by categorizing them into emotional clusters with a self-organizing layer. The proposed model is evaluated with three different corpora: The Surrey Audio–Visual Expressed Emotion (SAVEE) database, the visual Bi-modal Face and Body benchmark (FABO) database, and the multimodal corpus of the Emotion Recognition in the Wild (EmotiW) challenge. We use these corpora to evaluate the performance of the model to recognize emotional expressions, and compare it to state-of-the-art research.
Hospital services quality assessment: hospitals of Kerman University of Medical Sciences, as a tangible example of a developing country.

PURPOSE Although quality orientation is one of the main priorities of any progressive organization, quality evaluation in organizations providing services such as hospitals is one of the key challenges, because in this sector quality is determined by many intangible factors. Applying the service quality gap model is one of the common tools for quality evaluation in the service sector. This paper seeks to evaluate this issue. DESIGN/METHODOLOGY/APPROACH The present descriptive study was carried out through a cross-sectional method in 2008. The participants of this study were patients who had been referred to Kerman University of Medical Sciences hospitals. The sample comprised 385 patients, the data were collected by SERVQUAL as a standard questionnaire, and data analysis was carried out on 385 completed questionnaires. FINDINGS In all five dimensions of quality, a gap was observed between patients' perceptions and expectations as follows: Assurance: -1.28, Empathy: -1.36, Responsiveness: -1.80, Tangibles: -1.86 and Reliability: -1.69. A paired T-test showed that the differences between quality perceptions and expectations are significant (p value < 0.05). Based on the findings of this research, the hospitals in the study did not meet the expectations of patients and were unable to provide health care services according to patients' expectations. Hence rearranging the service delivery and deploying better facilities and equipment in order to decrease the gap between patients' perceptions and expectations may be helpful. ORIGINALITY/VALUE The paper reports an original application of the SERVQUAL approach to hospital services of a developing country.
A power electronic transformer based on a 4-plate compact capacitive coupler

This paper proposes a power electronic transformer based on a capacitive coupler. The coupler consists of four plates which are parallel-placed very closely. And there is a dielectric layer between each two plates to increase the capacitance value. The two plates on the same side are placed close to each other to maintain a large capacitance. The system topology is symmetrical to achieve bi-directional power transfer as both sides are composed of a full bridge inverter and a LCL circuit. The LCL circuit is used for filtering waves and forming resonance with the coupler. The coupler is modeled to simplify the analysis of the circuit. The whole system is verified by theory and simulation.
Occupational exposure of gasoline station workers to BTEX compounds in Bangkok, Thailand.

BACKGROUND Gasoline station workers are exposed to volatile organic compounds such as benzene, toluene, ethylbenzene and xylene (BTEX). OBJECTIVE To determine the level of exposure to BTEX compounds among gasoline station workers and measure the roadside concentrations of these compounds in the inner and outer areas of Bangkok, Thailand. METHODS 49 workers at 6 gasoline stations in the inner and outer areas of Bangkok participated in this study. Samples of ambient air were collected from the area near gas pumps at each station and at the roadside in front of the gas stations by charcoal tubes. All samples were analyzed for BTEX compounds by gas chromatography-flame ionized detector (GC-FID). RESULTS The mean BTEX concentration in gas stations was slightly higher than that of the roadside; there was no significant difference in the concentration between inner and outer areas. The mean lifetime cancer risks for workers exposed to benzene and ethylbenzene for 30 years were estimated at 1.75×10⁻⁴ and 9.55×10⁻⁷. The estimated hazard quotients for BTEX compounds were 0.600, 0.008, 0.007 and 0.002, respectively. The most prevalent symptoms of workers were headache (61%), fatigue (29%) and throat irritation (11%), respectively. Exposure to benzene and toluene was significantly associated with fatigue (p<0.05). CONCLUSION Exposure to BTEX compounds would increase the risk of cancer in gasoline station workers. Exposure to benzene and toluene may cause fatigue.
In vivo inhibition of the antibody response by a complement receptor- specific monoclonal antibody

BALB/c mice were injected intravenously with three different monoclonal antibodies (mAbs) specific for complement receptor 1 (CR1). Two of the mAbs crossreacted with CR2. 24 h later, the mice were immunized with horse erythrocytes or keyhole limpet hemocyanin (KLH), and the primary antibody response was measured. One of the anti-CR antibodies, 7G6, suppressed greater than 99% of the direct plaque-forming cell response against horse red blood cells (HRBC). The same antibody markedly suppressed the serum antibody responses to both HRBC and KLH. To be optimally suppressive, the mAb had to be injected before suboptimal concentrations of antigen. The other two complement receptor-specific antibodies had very moderate, if any, effects on the antibody response. 7G6 was able to downregulate CR1 and CR2 on the surface of B cells and, in addition, to inhibit rosette formation with C3d-coated sheep erythrocytes (EC3d). One of the antibodies with a weak effect downregulated only CR1. The other downregulated both CR1 and CR2, although not as efficiently as 7G6, and was unable to inhibit EC3d rosette formation. We conclude that the reason 7G6 is outstanding in its suppressive capacity is that it is the only mAb tested that functionally blocks CR2. The data suggest that CR2 is of crucial importance in the initiation of a normal antibody response to physiological concentrations of antigen.
Defining the ATM-mediated barrier to tumorigenesis in somatic mammary cells following ErbB2 activation

p53, apoptosis, and senescence are frequently activated in preneoplastic lesions and are barriers to progression to malignancy. These barriers have been suggested to result from an ATM-mediated DNA damage response (DDR), which may follow oncogene-induced hyperproliferation and ensuing DNA replication stress. To elucidate the currently untested role of DDR in breast cancer initiation, we examined the effect of oncogene expression in several murine models of breast cancer. We did not observe a detectable DDR in early hyperplastic lesions arising in transgenic mice expressing several different oncogenes. However, DDR signaling was strongly induced in preneoplastic lesions arising from individual mammary cells transduced in vivo by retroviruses expressing either PyMT or ErbB2. Thus, activation of an oncogene after normal tissue development causes a DDR. Furthermore, in this somatic ErbB2 tumor model, ATM, and thus DDR, is required for p53 stabilization, apoptosis, and senescence. In palpable tumors in this model, p53 stabilization and apoptosis are lost, but unexpectedly senescence remains in many tumor cells. Thus, this murine model fully recapitulates early DDR signaling; the eventual suppression of its endpoints in tumorigenesis provides compelling evidence that ErbB2-induced aberrant mammary cell proliferation leads to an ATM-mediated DDR that activates apoptosis and senescence, and at least the former must be overcome to progress to malignancy. This in vivo study also uncovers an unexpected effect of ErbB2 activation previously known for its prosurvival roles, and suggests that protection of the ATM-mediated DDR-p53 signaling pathway may be important in breast cancer prevention.
An online help framework for web applications

As the number of users using web-based applications increases, online help systems are required to provide appropriate information in multiple formats and accesses. Since users are dealing with various applications simultaneously so as to complete objectives and make daily decisions, it is necessary to provide anytime, anywhere help or tutorials. However, developing effective and efficient help systems on behalf of users is costly. As geographically distributed and multilingual users with minimal training or interruption have increased significantly, it has become a requirement to restructure help systems. In order to meet users' requirements and business challenges, it is necessary to restructure help systems to deliver training, documentation, and online help while taking into account nonfunctional requirements such as usability, time-to-market, quality and maintainability. In this paper we try to overcome such technical and economic constraints through single sourcing and content reuse, embrace as many users as possible by HTML-based help systems, and beef up help contents through unified and structural help system design. In addition, network pipe method for user feedback data collection and statistical analysis has been suggested as user feedback automation.
Targeting CCR2 or CD18 Inhibits Experimental In-Stent Restenosis in Primates: Inhibitory Potential Depends on Type of Injury and Leukocytes Targeted

A central role for leukocytes in neointimal hyperplasia after arterial injury is suspected. However, the relative importance of neutrophils and monocytes in balloon or stent-induced injury are not well understood, and mechanistic targeting of leukocyte recruitment or function is crude. We determined the temporal and spatial distribution of different leukocytes after balloon and stent-induced injury in primate iliac arteries. Based on these data, we targeted neutrophil and monocyte recruitment selectively after angioplasty or stent implantation and demonstrated that monocyte-specific blockade achieved via blockade of the MCP-1 receptor CCR2, was effective at reducing neointimal hyperplasia after stenting. In contrast, combined neutrophil and monocyte blockade achieved by targeting the leukocyte &bgr;2-integrin &bgr;-subunit CD18 was required to reduce neointimal hyperplasia after balloon injury. Distinct patterns of leukocyte infiltration in balloon versus stent-injured arteries predict distinct mechanisms for antiinflammatory strategies targeting neutrophils or monocytes in primates and may assist design of effective clinical strategies for optimizing vascular interventions.
A Theological and Psychological Defense of Self-Forgiveness: Implications for Counseling

In the burgeoning literature of forgiveness, self-forgiveness is an aspect that has not received as much attention as other areas of research on forgiveness. Self-forgiveness is important in dealing with negative feelings toward the self when one violates the conscience. However, because criticisms have emerged against this construct, we attempt a theological and psychological defense of self-forgiveness by addressing following four questions: (a) Is self-forgiveness the same as or different from divine forgiveness? (b) What is the nature of self-forgiveness? (c) Can we rationally address the criticisms of self-forgiveness? (d) How does self-forgiveness interact with divine and person-to-person forgiveness? After addressing these questions, we delineate four implications for counselors working with clients struggling to forgive themselves. Self-forgiveness, as one form of the virtue of forgiveness, now needs to be opened up more widely and deeply for more clients, and their emotional health may be further enhanced by this experience that frees them from self-condemnation when guided by a counselor who knows the essence of what self-forgiveness is and is not.