Miracle-dz/UniFAR-FTUs huggingface dataset:
Compensation Method for Occluded-region of Arbitrary-view Image Synthesized from Multi-view Video

In this paper, we propose a method for an arbitrary-view image generation in multi-view video and methods for pre- and post-processing to compensate unattended regions in the generated image. To generate an arbitrary-view image, camera geometry is used. Three dimensional coordinates of image pixels can be obtained by using depth information of multi-view video and parameter information of multi-view cameras, and by replacing three dimensional coordinates on a two dimensional image plane of other view, arbitrary-view image can be reconstructed. However, the generated arbitrary-view image contains many unattended regions. In this paper, we also proposed a method for compensating these regions considering temporal redundancy and spatial direction of an image and an error of acquired multi-view image and depth information. Test results show that we could obtain a reliably synthesized view-image with objective measurement of PSNR more than 30dB and subjective estimation of DSCQS(double stimulus continuous quality scale method) more than 3.5 point.
The Ground State Energy of a Dilute Bose Gas in Dimension n >3

We consider a Bose gas in spatial dimension $n>3$ with a repulsive, radially symmetric two-body potential $V$. In the limit of low density $\rho$, the ground state energy per particle in the thermodynamic limit is shown to be $(n-2)|\mathbb S^{n-1}|a^{n-2}\rho$, where $|\mathbb S^{n-1}|$ denotes the surface measure of the unit sphere in $\mathbb{R}^n$ and $a$ is the scattering length of $V$. Furthermore, for smooth and compactly supported two-body potentials, we derive upper bounds to the ground state energy with a correction term $(1+C\gamma)8\pi^4a^6\rho^2|\ln(a^4\rho)|$ in dimension $n=4$, where $\gamma:=\int V(x)|x|^{-2}\, dx$, and a correction term which is $\mathcal{O}(\rho^2)$ in higher dimensions.
Characterization of Two Newly Identiﬁed Genes, vgaD and vatG , Conferring Resistance to Streptogramin A in Enterococcus faecium (cid:1)

We characterized two new streptogramin A resistance genes from quinupristin-dalfopristin-resistant En- terococcus faecium JS79, which was selected from 79 E. faecium isolates lacking known genes encoding streptogramin A acetyltransferase. A 5,650-bp fragment of HindIII-digested plasmid DNA from E. faecium JS79 was cloned and sequenced. The fragment contained two open reading frames carrying resistance genes related to streptogramin A, namely, genes for an acetyltransferase and an ATP efﬂux pump. The ﬁrst open reading frame comprised 648 bp encoding 216 amino acids with a predicted left-handed parallel (cid:1) -helix domain structure; this new gene was designated vatG . The second open reading frame consisted of 1,575 bp encoding 525 amino acids with two predicted ATPase binding cassette transporters comprised of Walker A, Walker B, and LSSG motifs; this gene was designated vgaD . vgaD is located 65 bp upstream from vatG , was detected together with vatG in 12 of 179 quinupristin-dalfopristin-resistant E. faecium isolates, and was on the same plasmid. the 5.6-kb HindIII-digested fragment observed in JS79 was detected in nine vgaD - and vatG -containing E. faecium isolates by Southern hybridization. Therefore, it was expected that these two genes were strongly correlated with each other and that they may be composed of a transposon. Importantly, vgaD is the ﬁrst identiﬁed ABC transporter conferring resistance to streptogramin A in E. faecium . Pulsed-ﬁeld gel electrophoresis patterns and sequence types of vgaD - and vatG -containing E. faecium isolates differed for isolates from humans and nonhumans.
Partial GMP-CS-LBP Face Recognition using Image Subblocks

—Proposed face recognition in this paper is a block based approach. Gabor magnitude-phase centrally symmetric local binary pattern (GMP-CS-LBP) has been used to extract directional texture characteristics of face at different spatial frequencies. CS-LBP is applied on the sub-blocks of magnitude and phase responses of Gabor images. Sparse classifier is employed as local classifier to find the sub-blocks class labels. We have evaluated the performance of the proposed algorithm on AR and ORL databases. In real world scenarios, partial face images are available to recognize the identity of an unknown individual. By comparing the recognition accuracy on the recognition results of image sub-blocks, we find the location and size of the most effective face sub-region for identification. Moreover, fusion of image sub-blocks at decision level leads to significantly improved recognition accuracy.
Regulation of the Deubiquitinating Enzyme CYLD by I (cid:2) B Kinase Gamma-Dependent Phosphorylation†

Tumor suppressor CYLD is a deubiquitinating enzyme (DUB) that inhibits the ubiquitination of key signaling molecules, including tumor necrosis factor (TNF) receptor-associated factor 2 (TRAF2). However, how the function of CYLD is regulated remains unknown. Here we provide evidence that inducible phosphor- ylation of CYLD is an important mechanism of its regulation. Under normal conditions, CYLD dominantly suppresses the ubiquitination of TRAF2. In response to cellular stimuli, CYLD undergoes rapid and transient phosphorylation, which is required for signal-induced TRAF2 ubiquitination and activation of downstream signaling events. Interestingly, the CYLD phosphorylation requires I (cid:1) B kinase gamma (IKK (cid:2) ) and can be induced by IKK catalytic subunits. These ﬁndings suggest that CYLD serves as a novel target of IKK and that the site-speciﬁc phosphorylation of CYLD regulates its signaling function. retroviral HA-tagged GST–c-Jun(1–79) CYLD into pGEX4T-1 vector mutants generated by site- mutagenesis. the sequenced
Acoustic signature reduction, modulation and control

In the present study the problem of the ship signature is dealt with considering the main perturbation sources that have a relevance on the propeller induced noise and vibrations. Specifically the following aspects that cause major effect on the hydroacoustic and propulsive performance of a marine propeller will be discussed: (i) the effect of the inflow, (ii) the contribution of the wake evolution and breakdown mechanisms, (iii) the propeller-rudder/propeller-strut interaction in conventional and podded propulsors. The results presented in the paper are part of research activities carried out by INSEAN, most of them supported by the Italian Navy. In this framework INSEAN has developed and implemented a number of advanced experimental and numerical tools, pioneering sometimes, which have aimed at probing into the complex problem of the hydrodynamic and hydroacoustic performance of a marine propeller.
Keratocyte Apoptosis and Failure of Corneal Allografts you. Your matters

Background— Murine models of high-risk and low-risk corneal transplantation were used to determine the role of keratocyte apoptosis in the failure of orthotopic allogeneic corneal transplants. Materials and Methods— Normal (low-risk, low-rejecting) and inflamed/vascularized (high-risk, high-rejecting) BALB/c recipient beds received fully mismatched C57BL/6 corneal allografts. Apoptosis was detected in the corneal stroma at various time points using an in situ terminal deoxynucleotide tranferase-mediated dUTP nick-end labeling assay, and ex vivo via Western analysis for active caspase-3. Apoptosis was also measured in a (donor-type) C57BL/6 keratocyte cell line after stimulation of Fas or via use of various pro-inflammatory cytokines. Results— Significantly more apoptotic cells were present in the stroma of rapidly rejecting high-risk corneal allografts compared with low-risk grafts. Apoptotic cells were shown to be nearly uniformly CD45 − and hence of a non-hematopoetic lineage. Apoptosis, however, was not present in highly inflamed but ungrafted corneas. Apoptosis was induced in keratocytes in vitro by dual stimulation with agonistic Fas mAb and either interleukin-1 β or tumor necrosis factor- α . Conclusion— Apoptosis of resident non-bone marrow-derived fibroblastic cells of the corneal stroma is strongly correlated with the failure of corneal allografts, particularly in the highly inflamed microenvironment of the high-risk allograft.
The Common Cytokine Receptor (cid:103) Chain Plays an Essential Role in Regulating Lymphoid Homeostasis

vital for for each of these cytokines the common cytokine receptor (cid:103) chain ( (cid:103) c ), and it shown that (cid:103) c -deficient mice exhibit se-verely compromised development and responsiveness to IL-2, IL-4, and IL-7. Nevertheless, these mice exhibit an age-dependent accumulation of splenic CD4 (cid:49) T cells, the majority of which have a phenotype typical of memory/activated cells. When (cid:103) c -deficient mice were mated to DO11.10 T cell receptor (TCR) transgenic mice, only the T cells bearing endogenous TCRs had this phenotype, suggesting that its acquisition was TCR dependent. Not only do the CD4 (cid:49) T cells from (cid:103) c -deficient mice exhibit an activated phenotype and greatly incorporation of bromodeoxyuridine but, consistent with the lack of (cid:103) c -dependent survival signals, they also exhibit an augmented rate of apoptosis. However, because the CD4 (cid:49) T cells accumulate, it is clear that the rate of proliferation exceeds the rate of cell
Targeted Disruption of the ÃŸlIntegrili Gene in a Lymphoma Cell Line Greatly Reduces Metastatic Capacity

Integrins have been implicated in tumor metastasis. To investigate this, we generated ÃŸlintegrin-negative double knockout (DKO) mutants of the highly metastatic ESb murine T-lymphoma cell line. The in vivo growth capacity of the mutants, which had lost a4ÃŸland aftÃŸlexpression, was not altered, but their metastatic capacity was greatly reduced. Tail vein injection of IO4ESb and single-knockout cells led to death of all animals within 9-11 days. In contrast, only one-half of the animals injected with IO4DKO cells died, but much later, after 20-60 days. The other one-half remained disease-free for up to 100 days. Whereas ESb and single-knockout cells disseminated predominantly to liver and spleen, metastasis of DKO cells to these organs was rare, even after this prolonged period. Instead, skeletal muscles were invaded extensively. Metastatic capacity was largely restored in a DKO clone, which had been transfected with ÃŸl cDNA and expressed ÃŸlat similar levels as ESbcells. Weconclude that /! I integrins are essential for efficient liver and spleen colonization by the ESb lymphoma.
From Domo de Eŭropa Historio en Ekzilo to the museum of human-hunting: from scenography to scenographics in the work of Thomas Bellinck

ABSTRACT This article explores how Belgian theatre maker Thomas Bellinck turns the scenography of an exhibition into a critical framework to examine the politics of memory and representation at play on the European continent. In his projects, Bellinck plays with conventional modes of displaying and exhibiting to challenge the current course of the European Union (as in Domo de Eŭropa Historio en Ekzilo) and question how the EU copes with migratory movements (as in his long-term and ongoing project Simple as ABC). By exhibiting fictive artefacts through conventional modes of displaying (pedestals, display cases), or bringing these modes of display to the stage, Bellinck highlights the agency and critical potential of these ‘mere’ instruments in an exhibition. Insisting on the performativity of the infrastructure of an exhibition becomes a scenographic strategy in Bellinck’s work to mark the role of scenography in the epistemological process that comes with visiting an exhibition and the discourses an exhibition tries to convey.
New regime, new policies: research ethics development – a case study of Uzbekistan

ABSTRACT New regimes need to make crucial decisions about the continuity of public policy. The new incumbents will decide whether to continue the previous administration’s policies or make radical changes. The choice between continuity and change may favour the former in the early stages. However, the propensity to introduce new services or means of delivery may increase with time. In the Central Asian experience, the pattern of policy development reflects in part the process of gaining independence and the dominant autocratic regime type. The first change of leader presents a significant choice between continuity and change. This article examines Uzbekistan, focusing on a prominent area on the policy agenda: higher education. It investigates how the state and universities have faced issues in research ethics in the humanities and social sciences. The article examines the pattern of adoption, adaption and abstention in the policy process and demonstrates the predominance of the last option.
Effect of Polyamines Application on Reducing Chilling Injury Incidence in Okra Pod ( Abelmoschus esculentus ( Stored at Low Storage Temperature

Generally, immature okra pod is perishable and sensitive to chilling when exposed to temperature below 10 °C. Polyamines application has been claimed to be able to cope with low temperature stress due to their polycationic and antioxidant properties. In the present study, the effects of putrescine, spermidine and spermine on maintaining quality of stored okra pod were investigated. Immature okra pods were treated with putrescine, spermidine and spermine at two different concentrations ( 0. 5 and 1. 0 mM) with four replications per treatment. On the other hand, the control okra pods were only dipped in distilled water. All the pods were stored at 4 °C with 85 ± 5% relative humidity for 12 days. Results showed that the okra pods treated with putrescine at both concentrations were significantly lower in chilling injury (CI) incidence (46 to 56% ) and weight loss (51 to 68% ) than the control. While spermidine and spermine showed no differences with control after 8 storage days. Exogenous putrescine application resulted in a higher DPPH scavenging activity as well as antioxidant enzymes activity of catalase and peroxidase with respect to control after 12 days of storage. These responses could possibly be involved in chilling tolerance in okra pod during cold storage.
CESifo Working Paper Series PARETO-IMPROVING INTERGENERATIONAL TRANSFERS

In the presence of endogenous growth intergenerational transfer from the young to the old reduce per capita income growth and harm future generations. On the other hand, competitive equilibria are inefficient if externalities sustain long-run growth. This paper shows that if individuals retire in the last period of their life, the inefficiency of the market economy can be removed by an investment subsidy without making the current or future generations worse off only if coupled with intergenerational transfers from the young to the old.
Using Integrated and Efficient Clustering Concept for Large Scale WSN

: A flourishing topology control approach, which can extend the lifetime and increase the scalability for wireless sensor networks, is clustering. The respected criteria for clustering methodology are to select cluster heads with more residual energy and to rotate them periodically.At substantial movement areas, Sensors tend to rapidly exhaust their energy resources and die much sooner, leaving behind energy hole and network segmentation. In this paper, a co-ordinated and proficient clustering idea for vast scale remote sensor network, the Integrated Distributed Clustering Algorithm (IDCA) is proposed in view of three ideas. Initially, the aggregated information is forwarded from cluster head to the base station through cluster head of the followingsuperior layer with shortest route between the cluster heads. Secondly, cluster head is chosen in view of the clustering element, which is the mix of residual energy and the quantity of neighbors of a specific node inside a cluster. Thirdly, each cluster has a emergencypreventionnode, which does the capacity of cluster head when the cluster head neglects to steel away its work in some basicconditions. The key point of the proposed calculation is to achieve energy proficiency and to extend the network lifetime. The proposed dispersed clustering calculation is appeared differently in relation to the current clustering calculations LEACH and HEED. The proposed clustering calculation demonstrates advancement in Network strength, Network lifetime, Packet conveyance proportion and Link dependability End to end delay and Control overhead is significantly lessenedin accordance to the simulation results.
CVR-2018-935R1 Title: Circulating blood cells and extracellular vesicles in acute cardioprotection Short title: Circulating cells and vesicles in cardioprotection

During an ST-elevation myocardial infarction (STEMI), the myocardium undergoes a prolonged period of ischaemia. Reperfusion therapy is essential to minimize cardiac injury but can paradoxically cause further damage. Experimental procedures to limit ischaemia and reperfusion (IR) injury have tended to focus on the cardiomyocytes since they are crucial for cardiac function. However, there is increasing evidence that non-cardiomyocyte resident cells in the heart (as discussed in a separate review in this Spotlight series) as well as circulating cells and factors play important roles in this pathology. For example, erythrocytes, in addition to their main oxygen-ferrying role, can protect the heart from IR injury via the export of nitric oxide bioactivity. Platelets are well-known to be involved in haemostasis and thrombosis, but beyond these roles, they secrete numerous factors including sphingosine-1 phosphate (S1P), platelet activating factor (PAF) and cytokines that can all strongly influence the development of IR injury. This is particularly relevant given that most STEMI patients receive at least one type of platelet inhibitor. Moreover, there are large numbers of circulating vesicles in the blood, including microvesicles and exosomes, which can exert both beneficial and detrimental effects on IR injury. Some of these effects are mediated by the transfer of miRNA to the heart. Synthetic miRNA molecules may offer an alternative approach to limiting the response to IR injury. We discuss these and other circulating factors, focussing on potential therapeutic targets relevant to IR injury. Given the prevalence of co-morbidities such as diabetes in the target patient population, their influence will also be discussed. This article is part of a Cardiovascular Research Spotlight Issue entitled ‘Cardioprotection Beyond the Cardiomyocyte’, and emerged as part of the discussions of the European Union (EU)-CARDIOPROTECTION Cooperation in Science and Technology (COST) Action, CA16225.
SINGLE ELECTRON TRANSISTOR IN PURE SILICON

Title of dissertation: SINGLE ELECTRON TRANSISTOR IN PURE SILICON Binhui Hu, Doctor of Philosophy, 2009 Dissertation directed by: Professor Chia-Hung Yang, Department of Electrical and Computer Engineering As promising candidates for spin qubits, semiconductor quantum dots (QDs) have attracted tremendous research efforts. Currently most advanced progress is from GaAs QDs. Compared to GaAs, lateral QDs in silicon are expected to have a spin coherence time orders of magnitude longer, because Si has zero nuclear spin, and there is no hyperfine interaction between electron spins and nuclear spins. We have developed enhancement-mode metal-oxide-semiconductor (MOS) single electron transistors (SETs) using pure silicon wafers with a bi-layer gated configuration. In an MOS-SET, the top gate is used to induce a two-dimensional electron gas (2DEG), just as in an MOS field effect transistor. The side gates deplete the 2DEG into a QD and two point-contact channels; one connects the QD to the source reservoir, and the other connects the QD to the drain reservoir. We have systematically investigated the MOS-SETs at 4.2 K, and separately in a dilution refrigerator with a base temperature of 10 mK. The data show that there is an intrinsic QD in each point-contact channel due to the local potential fluctuations in these SETs. However, after scaling down the SETs, we have found that the intrinsic QDs can be removed and the electrostatically defined dots dominate the device behavior, but these devices currently only work in the many-electron regime. In order to realize single electron confinement, it is necessary to continue scaling down the device and improving the interface quality. To explore the spin dynamics in silicon, we have investigated a single intrinsic QD by applying a magnetic field perpendicular to the sample surface. The magnetic field dependence of the ground-state and excited-state energy levels of the QD mostly can be explained by the Zeeman effect, with no obvious orbital effect up to 9 T. The two-electron singlet-triplet (ST) transition is first time directly observed in a silicon QD by excitation spectroscopy. In this ST transition, electron-electron Coulomb interaction plays a significant role. The observed amplitude spectrum suggests the spin blockade effect. When the two-electron system forms a singlet state in the dot at low fields, and the injection current from the lead becomes spin-down polarized, the tunneling conductance is reduced by a factor of 8. At higher magnetic fields, due to the ST transition, the spin blockade effect is lifted and the conductance is
A pr 2 00 8 Analytic calculation of quasi-normal modes

We discuss the analytic calculation of quasi-normal modes o f various types of perturbations of black holes both in asymptotically flat and anti-de Sitter sp aces. We obtain asymptotic expressions and also show how corrections can be calculated perturbativ ely. We pay special attention to lowfrequency modes in anti-de Sitter space because they govern th hydrodynamic properties of a gauge theory fluid according to the AdS/CFT correspondence. The latter may have experimental consequencies for the quark-gluon plasma formed in heavy io n collisions. Prepared for the proceedings of the 4th Aegean Summer School on Black Holes, Mytilene, Greece, September 2007. Research supported in part by the US Department of Energy und er grant DE-FG05-91ER40627. †E-mail: siopsis@tennessee.edu
LYMPHOID NEOPLASIA Integrated phosphoproteomic and metabolomic pro ﬁ ling reveals NPM-ALK – mediated phosphorylation of PKM2 and metabolic reprogramming in anaplastic large cell lymphoma

The mechanisms underlying the pathogenesis of the constitutively active tyrosine kinase nucleophosmin-anaplastic lymphoma kinase (NPM-ALK) expressing anaplastic large cell lymphoma are not completely understood. Here we show using an integrated phosphoproteomic and metabolomic strategy that NPM-ALK induces a metabolic shift toward aerobic glycolysis, increased lactate production, and biomass production. The metabolic shift is mediated through the anaplastic lymphoma kinase (ALK) phosphorylation of the tumor-specific isoform of pyruvate kinase (PKM2) at Y105, resulting in decreased enzymatic activity. Small molecule activation of PKM2 or expression of Y105F PKM2 mutant leads to reversal of the metabolic switch with increased oxidative phosphorylation and reduced lactate production coincident with increased cell death, decreased colony formation, and reduced tumor growth in an in vivo xenograft model. This study provides comprehensive profiling of the phosphoproteomic and metabolomic consequences of NPM-ALK expression and reveals a novel role of ALK in the regulation of multiple components of cellular metabolism. Our studies show that PKM2 is a novel substrate of ALK and plays a critical role in mediating the metabolic shift toward biomass production and tumorigenesis. ( Blood . 2013;122(6):958-968)