Miracle-dz/UniFAR-FTUs huggingface dataset:
The Development of Shadow Play Wayang Kulit Using Augmented Reality

Augmented Reality (AR) is a current technology which creates a virtual object in a physical real world environment that creates a mixed reality. This paper describes the process of the development of an AR application augmented reality shadow play puppet- wayang kulit. Wayang Kulit is a traditional theatre that uses the principle of light and shadow accompanied with traditional music. Nowadays, the interest on wayang kulit among the young generation is slowly being fading out; efforts in terms of digitization would help to enlighten the valuable performing arts icon to the country. To preserve the country heritage, this study was conducted to introduce the art of shadow puppets in the form of application or a system that is easy for users to feel how to play wayang kulit. To create the application, the puppets are modeled in 3D graphic model and ARToolkit is selected as an application that overlays virtual imagery on the real world whereas Microsoft Visual C++ is used for developing the application. This application has a huge potential in promoting the wayang kulit and as an attraction as well as to promote the Wayang Kulit as one of the traditional art.
A Survey On Security Evaluation Of Pattern Classifiers Under Attack

Pattern classification is a branch of machine learning that focuses on recognition of patterns and regularities in data.In adversarial applications like biometric authentication, spam filtering, network intrusion detection the pattern classification systems are used. Pattern classification systems may exhibit vulnerabilities if adversarial scenario is not taken into account. Multimodal biometric systems are more robust to spoofing attacks,as they combine information coming from different biometric traits.In this paper,weevaluate the security of pattern classifiersthat formalizes and generalizes the main ideas proposed in the literature and give examples of its use in three real applications.We propose a framework for evaluation of pattern security,model of adversary for defining any attack scenario.Reported results show that security evaluation can provide a more complete understanding of the classifier's behavior in adversarial environments, and lead to better design choices.
Properties of Quisqualate‐Sensitive L‐[3H]Glutamate Binding Sites in Rat Brain as Determined by Quantitative Autoradiography

Abstract: Quisqualate, a glutamate analogue, displaced L‐[3H]glutamate binding in a biphasic manner, corresponding to “high‐affinity” and “low‐affinity” binding sites. High‐affinity quisqualate sites were termed “quisqualate‐sensitive L‐[3H]glutamate” binding sites. Quisqualate‐sen‐sitive L‐[3H]glutamate binding was regionally distributed, with the highest levels present in the cerebellar molecular layer. This binding was stimulated by millimolar concentrations of chloride and calcium. The stimulatory effects of calcium required the presence of chloride ions, whereas chloride's stimulatory effects did not require calcium. All of the L‐[3H]glutamate binding stimulated by chloride/calcium was quisqualate sensitive and only weakly displaced by N‐methyl‐D‐aspartate, L‐aspartate, or kainate. At high concentrations (1 mM), the anion blockers 4‐acetamido‐4′‐isothiocyanostilbene‐2,2′‐disulfonic acid and4,4′‐diisothio‐cyanatostilbene‐2,2′‐disulfonic acid both reduced, by 41 and 43%, respectively, the stimulatory effects of chloride. At concentrations of 100 μM, kynurenate, L‐aspartate, (RS)‐α‐amino‐3‐hydroxy‐5‐methyl‐4‐isoxazolepropionic acid (AMPA), and L‐2‐amino‐4‐phosphonobutyric acid (L‐APB) failed to displace quisqualate‐sensitive L‐[3H]‐glutamate binding in the cerebellar molecular layer. In the presence of KSCN, however, 100 μM AMPA displaced 44% of binding. Quisqualate‐sensitive L‐[3H]glutamate binding was not sensitive to freezing, and, in contrast to other chloride‐ and calcium‐dependent L‐[3H]glutamate binding sites that have been reported, quisqualate‐sensitive binding observed by autoradiography was enhanced at 4°C compared with 37°C. Quisqualate‐sensitive L‐[3H]glutamate binding likely represents binding to the subclass of postsynaptic neuronal glutamate receptors known as quisqualate receptors, rather than binding to previously described APB receptors, chloride‐driven sequestration into vesicles, or binding to astrocytic membrane binding sites.
Wireless LAN Comes of Age: Understanding the IEEE 802.11n Amendment

During the initial development of the IEEE 802.11n (11n) amendment for improving the throughput of wireless LANs, a lot of excitement existed surrounding the potential higher throughput (i.e., faster downloads), and increased range (distance) achievable. However, delays in the development of this standard (which began in 2003, and is still in the final draft stages) as well as vendor, customer reluctance to adopt the pre-11n offerings in the marketplace, have generally slowed interest in this next-generation technology. However, there is still much to be excited about. The latest draft of IEEE 802.11n (Draft 3.0) offers the potential of throughputs beyond 200 Mbps, based on physical layer (PHY) data rates up to 600 Mbps. This is achieved through the use of multiple transmit and receive antennas, referred to as MIMO (multiple input, multiple output). Using techniques such as spatial division multiplexing (SDM), transmitter beamforming, and space-time block coding (STBC), MIMO is used to increase dramatically throughput over single antenna systems (by two to four times) or to improve range of reception, depending on the environment. This article offers an exposition on the techniques used in IEEE 802.11n to achieve the above improvements to throughput and range. First, the current generation WLAN devices (11a/b/g) are described in terms of the benefits offered to end users. Next, the evolution of the Tin amendment is discussed, describing the main proposals given, and illustrating reasons for the delay in standardization. Then, the changes to the PHY for 11n are presented. A description of channel modeling with MIMO is shown, followed by the signal processing techniques employed, including MIMO channel estimation and detection, space-time block coding (STBC), and transmitter beamforming. Simulation results are presented which illustrate the benefits of these techniques, versus the existing a/g structures, for both throughput and range. Finally, a brief section outlining considerations for the rapid prototyping of a baseband design based on the 802.11n PHY is presented. We conclude with a discussion of the future for 11n, describing the issues addressed with Drafts 2.0 and 3.0, as well as its place in a wireless market with WiMAX and Bluetooth.
Regulation of virulence by BarA-UvrY two-component system and LuxS in extraintestinal pathogenic Escherichia coli

Title of the Thesis: Regulation of Virulence by BarA-UvrY TwoComponent system and LuxS in Extraintestinal Pathogenic Escherichia coli. Senthilkumar Palaniyandi, Masters, 2007 Directed By: Dr. Suman Mukhopadhyay, Assistant Professor, Department of Veterinary Medicine Pathogenic E. coli cause intestinal or extraintestinal infections in many host species. E. coli strains enter the intestinal tract through food and colonize the intestinal epithelium to cause infections. In animals and humans, E. coli causes gastroenteritis, neonatal meningitis and urinary tract infections. In birds, E. coli causes a complex syndrome called avian colibacillosis. The orthologs of BarA-UvrY two-component (TCS) system is known to regulate a number of phenotypic traits in gamma proteobacteria, although their role in Extraintestinal pathogenic Escherichia coli (ExPEC) virulence is yet to be determined. The barA gene is membrane bound sensor kinase protein and the uvrY gene encodes the cognate response regulator in E. coli. Work in this study has focused how the BarA-UvrY and LuxS system regulates in vivo virulence in uropathogenic E. coli (UPEC) and avian pathogenic E. coli (APEC) during infection. The main goal of this study is to look at how BarA-UvrY TCS and LuxS regulate virulence in APEC χ7122 and UPEC CFT073. In this study, we studied the role of BarA-UvrY TCS system in regulation of virulence in the aforementioned ExPEC strains using animal model and tissue culture system and the role of LuxS in regulation of virulence determination in ExPEC. Our results indicate that BarA-UvrY regulates multiple virulence properties in APEC χ7122 and UPEC CFT073 and that LuxS regulates partial virulence properties in APEC χ7122 and UPEC CFT073. REGULATION OF VIRULENCE BY BarA-UvrY TWOCOMPONENT SYSTEM AND LuxS IN EXTRAINTESTINAL PATHOGENIC ESCHERICHIA COLI.
Radial velocities of dwarf spheroidal galaxies in the M 81 group

Long-slit observations of 4 dwarf spheroidal galaxies in the M 81 group are presented. We have obtained the heliocentric velocity of a globular cluster candidate located near the center of DDO 78 to be 55 10 km s 1 by cross-correlation with template stars. We estimated a heliocentric radial velocity of 116 21 km s 1 for an HII region seen in K 61. A red diuse object near the K 64 center is found to be a remote galaxy with a heliocentric velocity of +46 530 km s 1 .
Heme oxygenase inhibition increases blood pressure in pregnant rats.

BACKGROUND During normal gestation, the placenta is a relatively hypoxic organ and, as such, is subject to significant oxidative stress. In the preeclamptic patient, inadequate remodeling of the maternal vasculature severely exacerbates placental oxidative stress, which has been shown to be an important component of maternal hypertension. There is emerging evidence that Heme Oxygenase-1 (HO-1) acts as an important regulator of placental and cardiovascular function during normal pregnancy. Here, we have examined the effect of Heme Oxygenase (HO) inhibition in late gestation on maternal blood pressure, angiogenic balance, and placental oxidative stress in pregnant rats. METHODS HO activity was inhibited with tin mesoporphyrin (SnMP), which was administered on gestational day 14, and blood pressure was measured on gestational day 19. Placental angiogenic balance and plasma Vascular Endothelial Growth Factor (VEGF) were determined by sandwich enzyme-linked immunosorbent assay. Nicotinamide adenine dinucleotide phosphate (NADPH) oxidase activity was measured by lucigenin chemilluminescence. RESULTS In response to SnMP treatment, maternal mean arterial pressure (MAP) was significantly increased (99±1 vs. 113±2mm Hg; P < 0.05; n = 15 per group). Placental soluble fms-like tyrosine kinase-1 (sFlt-1) (631±47 vs. 648±26 pg/mg; P = 0.76) levels in the placenta were not affected by HO inhibition. Additionally, there was no significant difference in free VEGF in the maternal circulation (287±22 vs. 329±14 pg/ml; P = 0.11). There was, however, a significant decrease in placental VEGF (23±2 vs. 16±1 pg/mg; P < 0.05) and a significant increase in placental NADPH oxidase activity in SnMP-treated rats (2021±238 vs. 3005±301 RLU/min/mg; P < 0.05). CONCLUSIONS Our results demonstrate that HO is an important regulator of blood pressure and an important antioxidant in the developing placenta.
THE CATEGORIZATION OF COMMON OBJECTS BY ADULTS WITH TRAUMATIC BRAIN INJURY: APPLICATION OF A SYSTEMATIC TRAINING PROGRAM

THE CATEGORIZATION OF COMMON OBJECTS BY ADULTS WITH TRAUMATIC BRAIN INJURY: APPLICATION OF A SYSTEMATIC TRAINING PROGRAM by Victoria L. Scharp This study investigated feature description abilities between normal subjects and subjects with traumatic brain injury across a continuum of stimulus modalities. Subjects were administered an extensive neuropsychological battery consisting of formal and informal measures prior to and following the completion of an eight level experimental Categorization Program. The objective of the present study was to analyze subject performance on the neuropsychological measures and the initial level (Level 1) of the Categorization Program. Overall, subjects with traumatic brain injury evidenced persistent deficits in information processing and cognitive flexibility; however, significant improvements were seen on functional outcome measures and informal tasks associated with the Categorization Program. Subjects with traumatic brain injury required more training trials in order to learn eight perceptual features and recalled fewer features per trial during the Feature Training Task, but they were able to apply the novel information presented and describe common objects effectively. The results of this study suggest that subjects with traumatic brain injury benefit from repeated presentations of information as it provides external structure and enhances working memory performance. THE CATEGORIZATION OF COMMON OBJECTS BY ADULTS WITH TRAUMATIC BRAIN INJURY: APPLICATION OF A SYSTEMATIC TRAINING PROGRAM A Thesis Submitted to Miami University in partial fulfillment of the requirements for the degree of Master of Arts Department of Speech Pathology and Audiology by Victoria L. Scharp Miami University Oxford, Ohio 2002 Advisor ____________________________ Fofi Constantinidou, Ph.D. Reader _____________________________ Robin D. Thomas, Ph.D. Reader _____________________________ Louise Van Vliet, Ph.D.
Participatory enthusiasms: a recent history of citizen engagement initiatives

ABSTRACT There have been waves of enthusiasm for citizen participation before. I discuss several features that make the current one distinctive: Its scale and scope, its reliance on modes of collaboration made familiar by digital media, its dependence on coalitions of actors, within and outside the state, for its effects, and its professionalization in techniques of facilitated discussion. I focus on the latter, exploring how professionals’ understandings of the aims and limits of participation sometimes conflict with those of participants. More broadly, I argue for a better understanding of how models of participation diffuse across institutional settings and with what effects.
Neuropsychological indicators of heteromodal cortex (dys)function relevant to conditioned blocking measures of attention in schizophrenia

Background. Learning a predictive relationship between two events can block learning about an added event (conditioned blocking; CB). Patients with nonparanoid schizophrenia can show reduced CB and learn about the similar consequences of the added event. Methods. This study reports on the relationship of associative learning and CB measures of attention to signs of heteromodal cortex function provided by performance on a battery of 10 neuropsychological tasks (i.e., frontal, parietal, and temporal lobe function of the left and right hemispheres). Acquisition criteria for the task were achieved by 62 patients with schizophrenia and 62 matched controls but not by 39 other individuals with schizophrenia. Results. First, right hemisphere, visuospatial abilities were generally associated with faster task learning, and patients who could not learn the task were poorer on tests emphasising set-switching abilities associated with left frontal lobe function. Second, CB expression depended on Stroop and Mooney faces task performance that are reported to require cingulate and parietal lobe function. Conclusions. As would be expected, right hemisphere function was implicated in performing a visuospatial learning task. The additional CB requirement incurred additional anterior cingulate and right parietal involvement. Functionally, this probably reflected effortful attentional processes, and illustrates the problems of patients with schizophrenia in switching between automatic and controlled processing strategies.
Depletion of the thiol oxidoreductase ERp57 in tumor cells inhibits proliferation and increases sensitivity to ionizing radiation and chemotherapeutics

Rapidly growing tumor cells must synthesize proteins at a high rate and therefore depend on an efficient folding and quality control system for nascent secretory proteins in the endoplasmic reticulum (ER). The ER resident thiol oxidoreductase ERp57 plays an important role in disulfide bond formation. Lentiviral, doxycycline-inducible ERp57 knockdown was combined with irradiation and treatment with chemotherapeutic agents. The knockdown of ERp57 significantly enhanced the apoptotic response to anticancer treatment in HCT116 colon cancer cells via a p53-dependent mechanism. Instead of a direct interaction with p53, depletion of ERp57 induced cell death via a selective activation of the PERK branch of the Unfolded Protein Response (UPR). In contrast, apoptosis was reduced in MDA-MB-231 breast cancer cells harboring mutant p53. Nevertheless, we observed a strong reduction of proliferation in response to ERp57 knockdown in both cell lines regardless of the p53 status. Depletion of ERp57 reduced the phosphorylation activity of the mTOR-complex1 (mTORC1) as demonstrated by reduction of p70S6K phosphorylation. Our data demonstrate that ERp57 is a promising target for anticancer therapy due to synergistic p53-dependent induction of apoptosis and p53-independent inhibition of proliferation.
A Critical and Cell-Autonomous Role for MeCP2 in Synaptic Scaling Up

Rett syndrome (Rett) is the leading genetic cause of mental retardation in females. Most cases of Rett are caused by loss-of-function mutations in the gene coding for the transcriptional regulator methyl-CpG binding protein 2 (MeCP2), but despite much effort, it remains unclear how a loss of MeCP2 function generates the neurological deficits of Rett. Here we show that MeCP2 plays an essential and cell-autonomous role in homeostatic synaptic scaling up in response to reduced firing or reduced sensory drive in rat visual cortical pyramidal neurons. We found that acute RNAi knockdown of MeCP2 blocked synaptic scaling within targeted neocortical pyramidal neurons. Furthermore, MeCP2 knockdown decreased excitatory synapse number without affecting basal mEPSC amplitude or AMPAR accumulation at spared synapses, demonstrating that MeCP2 acts cell-autonomously to maintain both excitatory synapse number and synaptic scaling in individual neocortical neurons. Finally, we used a mouse model of Rett to show that MeCP2 loss prevents homeostatic synaptic scaling up in response to visual deprivation in vivo, demonstrating for the first time that MeCP2 loss disrupts homeostatic plasticity within the intact developing neocortex. Our results establish MeCP2 as a critical mediator of synaptic scaling and raise the possibility that some of the neurological defects of Rett arise from a disruption of homeostatic plasticity.
A Primer on Using Behavioral Data for Testing Theories in Advertising Research

Interactions with and between customers in digital, social, and mobile environments are commonly recorded, producing behavioral data that have the potential to advance advertising research. This article provides an accessible guide on how to leverage such data for advertising researchers who may have thus far relied mostly on lab experiment or survey data. Specifically, we suggest potential sources for behavioral data and present a process for analyzing and interpreting behavioral data. Each step of the process is discussed: exploring, understanding and preparing data; specifying and estimating models; and interpreting and presenting the results. Some fundamental issues with using multiple regression to analyze such data are covered, including standardization, outliers, transformations, multicollinearity, and the omitted variable bias. We also discuss issues that are especially problematic with using behavioral data in advertising research, including endogeneity, count data, data with many zeros, and grouped data. More advanced versions of regression that address these issues are surveyed, including instrumental variables, propensity scoring, generalized linear models, and mixed models. General advice for thinking about behavioral data is provided.
Coordination of upper-limb movement : a neuro-dynamics account

Human performers are often limited in doing more than one thing at a time. This becomes particularly evident when different movements are to be performed simultaneously. The emerging limitations result from a tendency to synchronize the patterns of motor output, irrespective of whether the movements are of a discrete or continuous nature. However, for differentiated patterns of activity to emerge, mutual synchronization has to be overcome. This paper addresses limitations in discrete dual-task performance and describes dissociation of action with respect to structural and metrical specifications of movement. Moreover, the role of augmented information feedback sources on interlimb dissociation is discussed
Advances in Swine Transcriptomics

The past five years have seen a tremendous rise in porcine transcriptomic data. Available porcine Expressed Sequence Tags (ESTs) have expanded greatly, with over 623,000 ESTs deposited in Genbank. ESTs have been used to expand the pig-human comparative maps, but such data has also been used in many ways to understand pig gene expression. Several methods have been used to identify genes differentially expressed (DE) in specific tissues or cell types under different treatments. These include open screening methods such as suppression subtractive hybridization, differential display, serial analysis of gene expression, and EST sequence frequency, as well as closed methods that measure expression of a defined set of sequences such as hybridization to membrane arrays and microarrays. The use of microarrays to begin large-scale transcriptome analysis has been recently reported, using either specialized or broad-coverage arrays. This review covers published results using the above techniques in the pig, as well as unpublished data provided by the research community, and reports on unpublished Affymetrix data from our group. Published and unpublished bioinformatics efforts are discussed, including recent work by our group to integrate two broad-coverage microarray platforms. We conclude by predicting experiments that will become possible with new anticipated tools and data, including the porcine genome sequence. We emphasize that the need for bioinformatics infrastructure to efficiently store and analyze the expanding amounts of gene expression data is critical, and that this deficit has emerged as a limiting factor for acceleration of genomic understanding in the pig.
WHY HAVE SOME INDIAN STATES PERFORMED BETTER THAN OTHERS AT REDUCING RURAL POVERTY ?

Rural poverty rankings of Indian states in 1990 were very different from 1960. This unevenness in progress allows us to study the causes of poverty in a developing rural economy. We model the evolution of various poverty measures, using pooled state-level data for the period 1957-91. Differences in trend rates of poverty reduction are attributed to differing growth rates of farm yield per acre, and differing initial conditions; states starting with better infrastructure and human resources saw significantly higher long-term rates of poverty reduction. Deviations from the trend are attributed to inflation (which hurt the poor in the short term) and shocks to farm and nonfarm output. CONTENTS Acknowledgments . v