Miracle-dz/UniFAR-FTUs huggingface dataset:
The moderating effects of demographic characteristics and certain psychological factors on the job embeddedness – turnover relationship among Thai health-care employees

This paper aims to investigate the roles of demographic characteristics (i.e. generations and organizational tenure) and psychological factors (i.e. leader-member exchange and self-efficacy) as moderators of the relationship between job embeddedness and turnover intention, and the mediating effect of turnover intention between job embeddedness and actual turnover. ,Data were collected from 422 health-care workers through a questionnaire survey and analyzed by means of a confirmatory factor analysis and hierarchical regression. ,The results reveal that less embedded employees who perceive a lower level of leader–member exchange quality are more likely to indicate an intention to leave. The negative relationship between job embeddedness and turnover intention is stronger among less embedded employees with high self-efficacy. The finding also indicates that turnover intention plays a significant mediating role in the relationship between job embeddedness and actual turnover. ,The current research took place within two health-care organizations. Replicating the study in a variety of industries, professions or cultures would be useful for the generalizability of the findings. ,Organizations may improve their retention of employees by nurturing the leader–member exchange relationship to enhance a social web that bonds them together. Managers may need to pay attention to making a greater effort to embed individuals in their jobs, so that they are better able to cope successfully with challenges and organize the workday to accommodate them. ,This study examines the moderating roles of individual characteristics and psychological factors on the relationship between job embeddedness and turnover intention, which has not been extensively investigated in the literature.
Decreased cortical representation of genital somatosensory field after childhood sexual abuse.

OBJECTIVE Sexual dysfunction is a common clinical symptom in women who were victims of childhood sexual abuse. The precise mechanism that mediates this association remains poorly understood. The authors evaluated the relationship between the experience of childhood abuse and neuroplastic thinning of cortical fields, depending on the nature of the abusive experience. METHOD The authors used MRI-based cortical thickness analysis in 51 medically healthy adult women to test whether different forms of childhood abuse were associated with cortical thinning in areas critical to the perception and processing of specific behavior implicated in the type of abuse. RESULTS Exposure to childhood sexual abuse was specifically associated with pronounced cortical thinning in the genital representation field of the primary somatosensory cortex. In contrast, emotional abuse was associated with cortical thinning in regions relevant to self-awareness and self-evaluation. CONCLUSIONS Neural plasticity during development appears to result in cortical adaptation that may shield a child from the sensory processing of the specific abusive experience by altering cortical representation fields in a regionally highly specific manner. Such plastic reorganization may be protective for the child living under abusive conditions, but it may underlie the development of behavioral problems, such as sexual dysfunction, later in life.
Care-full failure: how auxiliary assistance to poor Roma migrant women in Spain compounds marginalization

ABSTRACT In Spain, the national and local authorities boast in recent years about their progressive programs for the integration of Roma migrants from Romania. Many state efforts to work with Roma on their integration are specifically directed at women. Economic integration into the waged labor market is considered a major goal as it, supposedly, leads to the empowerment of Roma migrant women while also securing decent standards of living for entire families. This article argues that integration programs adversely result in the further discrimination and exclusion of those they pretend to relief. This adverse result is produced through a two-tier intervention in the lives of Roma families. The caring state works with a general category of ‘vulnerability’ for targeting populations, in which Roma migrant women are specifically incorporated through designated social programs. The performance of Roma as the subject–object of these programs is carefully evaluated. According to these evaluations, Roma women often fail to meet the normative standards of ‘good mothers’, ‘decent wives’, and ‘diligent workers’. Subsequently, to deal with ‘failing subjects’, the disciplining state, a-la Foucault, inflicts an array of penalties on Roma women and their families: cut-offs of social benefits, evictions from poor dwellings, withdrawal of children’s custody, and forced removals to Romania. We thus argue that initiatives by the caring state (and civil society) often prescribe or go hand-in-hand with repression from the correcting state. In welfare states, social programs can thus conclusively ‘evidence’ existing stereotypes about marginalized Roma families and about women in particular.
Effect of Polyvinyl Alcohol (PVA) Containing Artemether in Treatment of Cutaneous Leishmaniasis Caused by Leishmania major in BALB/c Mice

Background: Polyvinyl alcohol (PVA) is one of the well-known polymers, which has been used in numerous biomedical applications because of its good biocompatibility. Objectives: Due to problems made by the therapeutics already used for leishmaniasis, the aim of this study was to evaluate the effect of PVA containing artemether in treating cutaneous leishmaniasis in BALB/c mice. Materials and Methods: Aqueous solution of PVA was prepared by mixing with Double Distilled Water. After preparation of PVA, 4.33 mg of each drug (main drug artemether and control drug 14% glucantime) was added to 100 g of prepared PVA-honey solution. The solution was incubated at 37°C and the release of artemether was evaluated by measuring absorbance at 260 nm wave length. In this study for treatment of mice lesion, we used PVA containing artemether and glucantime and this method was compared with ointment treatment. Results: Mean diameters of lesions in mice treated with artemether were smaller than the control group and the differences were significant (P < 0.05). The mean lesion size of mice treated with PVA containing artemether in comparison with the group treated with ointment of artemether were smaller and the differences were significant (P < 0.05). Conclusions: PVA containing artemether is a new method for treatment of cutaneous leishmaniasis and according to the obtained results, artemether is an appropriate and effective drug, especially when used with PVA as a lesion dressing; thus we suggest that this method can be applied for the treatment of cutaneous leishmaniasis.
ARF-GEF cytohesin-2/ARNO regulates R-Ras and α5-integrin recycling through an EHD1-positive compartment

R-Ras and cytohesin-2/ARNO coordinate in the control of epithelial cell adhesion, but the mechanism has been unclear. Cytohesin-2/ARNO regulates traffic through an EHD1-positive recycling compartment. Inhibition of cytohesin-2/ARNO activity traps R-Ras and integrins within the EHD1 compartment and impairs adhesion and spreading.
Effect of Motor Imagery in Children with Unilateral Cerebral Palsy: fMRI Study

Background Motor imagery is considered as a promising therapeutic tool for rehabilitation of motor planning problems in patients with cerebral palsy. However motor planning problems may lead to poor motor imagery ability. Aim The aim of this functional magnetic resonance imaging study was to examine and compare brain activation following motor imagery tasks in patients with hemiplegic cerebral palsy with left or right early brain lesions. We tested also the influence of the side of imagined hand movement. Method Twenty patients with clinical hemiplegic cerebral palsy (sixteen males, mean age 12 years and 10 months, aged 6 years 10 months to 20 years 10 months) participated in this study. Using block design, brain activations following motor imagery of a simple opening-closing hand movement performed by either the paretic or nonparetic hand was examined. Results During motor imagery tasks, patients with early right brain damages activated bilateral fronto-parietal network that comprise most of the nodes of the network well described in healthy subjects. Inversely, in patients with left early brain lesion brain activation following motor imagery tasks was reduced, compared to patients with right brain lesions. We found also a weak influence of the side of imagined hand movement. Conclusion Decreased activations following motor imagery in patients with right unilateral cerebral palsy highlight the dominance of the left hemisphere during motor imagery tasks. This study gives neuronal substrate to propose motor imagery tasks in unilateral cerebral palsy rehabilitation at least for patients with right brain lesions.
Further Opportunities to Reduce the Energy Use and Greenhouse Gas Emissions of Buildings

Buildings and energy systems are shaped within many different kinds of departments and agencies throughout local governments. This article argues that further opportunities exist to reduce the energy use of buildings and their associated greenhouse gas (GHG) emissions through the existing powers and jurisdiction of local governments. We use the example of New York City, where buildings produced 73 percent of all GHG emissions in 2014. By analyzing a data set of almost four thousand large buildings, we identify new opportunities for planners and other professionals to reduce energy use and GHG emissions by focusing on different mechanisms and/or collaborations.
Cbln3, a Novel Member of the Precerebellin Family that Binds Specifically to Cbln1

Precerebellin (Cbln1) is the precursor of the brain-specific hexadecapeptide cerebellin. Although cerebellin has properties of a conventional neuropeptide, its function is controversial because Cbln1 has structural features characteristic of circulating atypical collagens. Cbln1 is related to the three subunits of the complement C1q complex. Therefore, we hypothesized that Cbln1 participated in analogous heteromeric complexes with precerebellin-related proteins. Using LexA-Cbln1 as bait in a yeast two-hybrid screen, we isolated a cDNA encoding a novel Cbln1-related protein, designated Cbln3. The gene encoding cbln3 had the same intron–exon structure ascbln1 but mapped to a different mouse chromosome (14). The deduced amino acid sequence of Cbln3 was 55% identical to Cbln1 and also contained a C1q signature domain and signal sequence for secretion. In addition to binding avidly to Cbln3, Cbln1 also formed homomeric complexes. In contrast, Cbln3 homomeric association was weak. These interactions exhibited specificity because C1qB bound to neither Cbln1 nor Cbln3. Like cbln1, cbln3 was expressed in the cerebellum and dorsal cochlear nucleus in which it was detected in granule neurons. Because Cbln1 and Cbln3 are coexpressed in the brain and interact avidly, they may function as a secreted heteromeric complex in vivo.
Salicylic acid is an indispensable component of the Ny-1 resistance-gene-mediated response against Potato virus Y infection in potato

The purpose of the study was to investigate the role of salicylic acid (SA) signalling in Ny-1-mediated hypersensitive resistance (HR) of potato (Solanum tuberosum L.) to Potato virus Y (PVY). The responses of the Ny-1 allele in the Rywal potato cultivar and transgenic NahG-Rywal potato plants that do not accumulate SA were characterized at the cytological, biochemical, transcriptome, and proteome levels. Analysis of noninoculated and inoculated leaves revealed that HR lesions started to develop from 3 d post inoculation and completely restricted the virus spread. At the cytological level, features of programmed cell death in combination with reactive oxygen species burst were observed. In response to PVY infection, SA was synthesized de novo. The lack of SA accumulation in the NahG plants led to the disease phenotype due to unrestricted viral spreading. Grafting experiments show that SA has a critical role in the inhibition of PVY spreading in parenchymal tissue, but not in vascular veins. The whole transcriptome analysis confirmed the central role of SA in orchestrating Ny-1-mediated responses and showed that the absence of SA leads to significant changes at the transcriptome level, including a delay in activation of expression of genes known to participate in defence responses. Moreover, perturbations in the expression of hormonal signalling genes were detected, shown as a switch from SA to jasmonic acid/ethylene signalling. Viral multiplication in the NahG plants was accompanied by downregulation of photosynthesis genes and activation of multiple energy-producing pathways.
Extracellular Calcium Regulates Postsynaptic Efficacy through Group 1 Metabotropic Glutamate Receptors

Bursts of synaptic transmission are known to induce transient depletion of Ca2+ within the synaptic cleft. Although Ca2+ depletion has been shown to lower presynaptic release probability, effects on the postsynaptic cell have not been reported. In this study, we show that physiologically relevant reductions in extracellular Ca2+ lead to a decrease in synaptic strength between synaptically coupled layer 2/3 cortical pyramidal neurons. Using quantal analysis and mEPSP analysis, we demonstrate that a lowered extracellular Ca2+ produces a reduction in the postsynaptic quantal size in addition to its known effect on release probability. An elevated Mg2+ level can prevent this reduction in postsynaptic efficacy at subphysiological Ca2+ levels. We show that the calcium-dependent effect on postsynaptic quantal size is mediated by group 1 metabotropic glutamate receptors, acting via CaMKII (Ca2+/calmodulin-dependent protein kinase II) and PKC. Therefore, physiologically relevant changes in extracellular Ca2+ can regulate information transfer at cortical synapses via both presynaptic and postsynaptic mechanisms.
Dense Correspondences across Scenes and Scales

We seek a practical method for establishing dense correspondences between two images with similar content, but possibly different 3D scenes. One of the challenges in designing such a system is the local scale differences of objects appearing in the two images. Previous methods often considered only few image pixels; matching only pixels for which stable scales may be reliably estimated. Recently, others have considered dense correspondences, but with substantial costs associated with generating, storing and matching scale invariant descriptors. Our work is motivated by the observation that pixels in the image have contexts-the pixels around them-which may be exploited in order to reliably estimate local scales. We make the following contributions. (i) We show that scales estimated in sparse interest points may be propagated to neighboring pixels where this information cannot be reliably determined. Doing so allows scale invariant descriptors to be extracted anywhere in the image. (ii) We explore three means for propagating this information: using the scales at detected interest points, using the underlying image information to guide scale propagation in each image separately, and using both images together. Finally, (iii), we provide extensive qualitative and quantitative results, demonstrating that scale propagation allows for accurate dense correspondences to be obtained even between very different images, with little computational costs beyond those required by existing methods.
Mechanisms of cognitive reserve : computational and experimental explorations

Cognitive reserve is the name given to the latent variable that describes individual differences in the ability to offset cognitive decline in old age. This thesis attempts to provide mechanistic explanations for two major aspects of cognitive reserve. These are neural compensation and neural reserve. Furthermore, behavioural experiments carried out as part of this investigation have extended the knowledge of existing theories as to the age invariance of neural compensation and the relationship between language, other more traditional proxies of cognitive reserve, and executive control. The results of these studies carried out in this thesis have demonstrated a biologically viable mechanism for the monitoring of task demand with resultant control of interhemispheric communication as a method of compensation. Further, this aspect of neural compensation was not found in younger participants. The neural network model in this thesis demonstrated differences over age in the spacing of representations for bilingual and monolingual networks as well as demonstrating increased inhibition in the bilingual network as a result of a negative relationship between weights from the tags of each language to nodes in the hidden layer. Finally, regression analysis using data from two large scale behavioural experiments demonstrated a minimal influence of bilingual language use on performance in executive control tasks. The models in this thesis provide an insight into the mechanisms behind cognitive reserve whilst supporting empirical results. Further, the results from the neural network model allowed predictions to be made with regard to the performance of bilinguals in dual category retrieval tasks. The lack of a relationship between bilingualism and cognitive control is supported by emerging research in the area and suggests that the functionality underlying cognitive reserve may be better described by biological rather than cognitive processes.
PersonNet: Person Re-identification with Deep Convolutional Neural Networks

In this paper, we propose a deep end-to-end neu- ral network to simultaneously learn high-level features and a corresponding similarity metric for person re-identification. The network takes a pair of raw RGB images as input, and outputs a similarity value indicating whether the two input images depict the same person. A layer of computing neighborhood range differences across two input images is employed to capture local relationship between patches. This operation is to seek a robust feature from input images. By increasing the depth to 10 weight layers and using very small (3$\times$3) convolution filters, our architecture achieves a remarkable improvement on the prior-art configurations. Meanwhile, an adaptive Root- Mean-Square (RMSProp) gradient decent algorithm is integrated into our architecture, which is beneficial to deep nets. Our method consistently outperforms state-of-the-art on two large datasets (CUHK03 and Market-1501), and a medium-sized data set (CUHK01).
Nuclear factor I gene expression in the developing forebrain

Three members of the Nuclear Factor I (Nfi) gene family of transcription factors; Nfia, Nfib, and Nfix are highly expressed in the developing mouse brain. Nfia and Nfib knockout mice display profound defects in the development of midline glial populations and the development of forebrain commissures (das Neves et al. [ 1999 ] Proc Natl Acad Sci U S A 96:11946–11951; Shu et al. [ 2003 ] J Neurosci 23:203–212; Steele‐Perkins et al. [ 2005 ] Mol Cell Biol 25:685–698). These findings suggest that Nfi genes may regulate the substrate over which the commissural axons grow to reach targets in the contralateral hemisphere. However, these genes are also expressed in the cerebral cortex and, thus, it is important to assess whether this expression correlates with a cell‐autonomous role in cortical development. Here we detail the protein expression of NFIA and NFIB during embryonic and postnatal mouse forebrain development. We find that both NFIA and NFIB are expressed in the deep cortical layers and subplate prenatally and display dynamic expression patterns postnatally. Both genes are also highly expressed in the developing hippocampus and in the diencephalon. We also find that principally neither NFIA nor NFIB are expressed in callosally projecting neurons postnatally, emphasizing the role for midline glial cell populations in commissure formation. However, a large proportion of laterally projecting neurons express both NFIA and NFIB, indicating a possible cell‐autonomous role for these transcription factors in corticospinal neuron development. Collectively, these data suggest that, in addition to regulating the formation of axon guidance substrates, these genes also have cell‐autonomous roles in cortical development. J. Comp. Neurol. 508:385–401, 2008. © 2008 Wiley‐Liss, Inc.
Journal of Personality and Social Psychology Absorption and Hypnotizability: Context Effects Reexamined

Two independent studies failed to find evidence consistent with Council, Kirsch, and Hafner (1986), who argued that the repeatedly observed correlations between Tellegen's (1981) Absorption Scale (TAS) and hypnosis measures were artifacts of testing context, and de Groot, Gwynn, and Spanos (1988), who claimed evidence for a Gender x Context moderator effect. In the present studies, Ss completed the TAS and other personality questionnaires on 2 occasions: during an independent survey and later immediately prior to an assessment of hypnotizability. In Experiment 1 (m = 475), the effect of context on the relation between questionnaire scores and hypnotizability was weak and variable; in Experiment 2 (N = 434), these weak effects were reversed. The results reat~rm the construct validity of absorption as both a major dimension of personality and as a predictor of hypnotic responsiveness. Beginning in the 1960s, several independent lines of psycho-metric work began to converge on a dimension of personality variously construed as "absorption" (Teilegen, 1981; Tellegen & Atkinson, 1974) and "openness to experience" (Coan, 1972; McCrae & Costa, 1985). A major impetus for this work was the attempt to discover personality correlates of individual differences in response to hypnosis (for reviews, see E. Although hypno-tizability did not appear to correlate with the dimensions measured on the common personality inventories such as the Min-number of separate investigations indicated that it did correlate with scales intended to measure imaginative involvement and other hypnotic-like experiences (e. Cohen for their good counsel on the statistical analyses and to Auke Tellegen for fruitful discussions concerning theoretical and statistical questions. 144 gested that the item pools of the most commonly used personality inventories did not in fact sample all aspects of personality and that significant additions would have to be made before they could be used to predict hypnotic response and similar aspects of psychological functioning. This suggestion gained credence with a factor analytic study by Tellegen and Atkinson (1974; see also Tellegen, 1987), who drew on the work of As, to develop a scale of absorption , defined as the subject's "full commitment of available perceptual , motoric, imaginative, and ideational resources to a unified representation of the attentional object" (p. 274). t Their study also included items representative of Block's (1965) factors of ego-control (extraversion) and ego-resiliency (neurotic-ism), the two major factors that run through all personality inventories. Absorption correlated significantly with hypno-tizability, but neither dimension correlated with either ego-control …
Induction of Disease Description based on Rough Sets

|This paper describes relations between rough set theory and rule-based description of neurological diseases, which corresponds to the process of knowledge acquisition of medical experts, and introduces automated induction of the rule-based description. The experimental results not only show that rough set theory gives a very suitable framework to represent processes of uncertain knowledge extraction, but also that this method induces diagnostic rules correctly