Model: OpenMed/OpenMed-ZeroShot-NER-Oncology-Tiny-60M
Author: OpenMed
Pipeline: token-classification
Library: gliner
Tags: gliner, pytorch, token-classification, entity recognition, named-entity-recognition, zero-shot, zero-shot-ner, zero shot, biomedical-nlp, cancer-genetics, oncology, gene-regulation, cancer-research, amino_acid, anatomical_system, cancer, cell, cellular_component, developing_anatomical_structure, gene_or_gene_product, immaterial_anatomical_entity, multi-tissue_structure, organ, organism, organism_subdivision, organism_substance, pathological_formation, simple_chemical, tissue, en, arxiv:2508.01630, license:apache-2.0, region:us
Card metadata: widget: Mutations in KRAS gene drive oncogenic transformation., The tumor suppressor p53 pathway was disrupted., EGFR amplification promotes cancer cell proliferation., Loss of function of the PTEN gene is common in many cancers., The PI3K/AKT/mTOR pathway is a critical regulator of cell growth.
tags: token-classification, entity recognition, named-entity-recognition, zero-shot, zero-shot-ner, zero shot, biomedical-nlp, gliner, cancer-genetics, oncology, gene-regulation, cancer-research, amino_acid, anatomical_system, cancer, cell, cellular_component, developing_anatomical_structure, gene_or_gene_product, immaterial_anatomical_entity, multi-tissue_structure, organ, organism, organism_subdivision, organism_substance, pathological_formation, simple_chemical, tissue
language: en
license: apache-2.0
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Created: 2025-09-15 21:14:11