Conversely, many SSRIs (but not fluoxetine, which is a 5-HT2C antagonist) indirectly stimulate 5-HT2C activity by increasing levels of serotonin in the synapse although the delayed mood elevation that's usually typical of SSRIs is usually paralleled by the downregulation of the 5-HT2C receptors.
Conversely, many SSRIs (but not fluoxetine, which is a 5-HT2C antagonist) indirectly stimulate 5-HT2C activity by increasing levels of serotonin in the synapse although the delayed mood elevation that’s usually typical of SSRIs is usually paralleled by the downregulation of the 5-HT2C receptors.