HDTenEightyP/NetNews huggingface dataset:
From: sdavis@khan.ColoSpgs.NCR.COM (Susan Davis)
Newsgroups: sci.med
Subject: Is there an FAQ for this group
Date: 15 Dec 92 21:57:14 GMT
Reply-To: sdavis@khan.ColoSpgs.NCR.COM (Susan Davis)
Distribution: world
Organization: NCR Microelectronic Products Division

I just started looking at this group and was wondering if there is an FAQ
available??  I am interested in an FAQ on migraine headaches.

Thanks,

Susan
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|Susan P. Davis, Design Services |  >>>>                 Susan.Davis@ncr-mpd |  |NCR Microelectronics  (AT&T)    | >>>>         Susan.Davis@ColoSpgs.NCR.COM |  |Colorado Springs, CO            |>>>>(719) 596-5795 x435 VOICEplus 463-0435 |
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From: dyer@spdcc.com (Steve Dyer)
Newsgroups: sci.med
Subject: Re: Peter Breggin, _Toxic Psychiatry_ (Q)
Date: 17 Dec 92 03:25:45 GMT
Article-I.D.: spdcc.1992Dec17.032545.22504
Organization: S.P. Dyer Computer Consulting, Cambridge MA

In article <BzD6GI.DvH@unx.sas.com> sasghm@theseus.unx.sas.com (Gary Merrill) writes:
>Yes, and you have put your finger precisely on the fundamental issue.
>It *doesn't matter* in such a case precisely what the disease is.
>There *is* a disease, and what is *important* is identifying the
>*cause* since it is *this* that determines treatment.

I would say that Toxic Shock Syndrome and a superficial staph infection
would be treated somewhat differently.  The fact that the same antibiotic
might be given to treat them both is neither here nor there, nor does it
alone constitute the "treatment".  What's more, the "causes" of many diseases
are not understood, and therefore have no bearing on their treatment.

>Again, what is the *analog* for schizophrenia?  I still have no
>answer to this.

So?  Who cares?

>The success of the disease model depends upon this.
>And upon the disease model rests much of contemporary psychiatry.

You have a pretty peculiar idea of what constitutes a "disease".
Contemporary psychiatry will sleep soundly.

>You are also correct that I do not recognize that schizophrenia
>is a real disease.  This is *not* "obviously incorrect" since
>in fact there is some long-standing dispute in the literature.

Not by anyone who knows what they're talking about.

>I deny only the cogency of the idea that there is a
>single identifiable disease under which all of this can be subsumed.
>Characterizing schizophrenia as a disease is not done by *subsuming*
>it under the existing disease model, but rather by *extending* that
>model intentionally to encompass a new domain.

Nonsense.  You don't even have the faintest idea of what a "disease" is,
let alone a "disease model".  How do you expect to argue against it?

>We are free to do
>this, but it results in changing what "disease" means and we must
>beware of being misled by a model that doesn't fit very well.

No it doesn't.  This is all consonant with its use elsewhere.

>There are several examples in the history of science where it is
>plain that clinging to such an incorrect model misdirected research
>and resulted in delaying advances.

Dyer's Law: The pomposity of a USENET posting is inversely proportional
to the knowledge and experience of its author.  Anyone care to try for
a corollary?

>It is clear that you consider schizophrenia to be a disease.  I
>wonder how you feel about such things as depression and attention
>deficit disorder.  Are these diseases as well?

Here we go again.  Anyone who has either experienced or witnessed major
depression or bipolar affective disorder wouldn't be asking such a
question.  Of course they're diseases.  There are objective criteria
which must be satisfied for a diagnosis.

>By the way, I don't mind your considering me ignorant.

"...Patient seems more in contact with reality in this last paragraph
than in previous days..."

-- 
Steve Dyer
dyer@ursa-major.spdcc.com aka {ima,harvard,rayssd,linus,m2c}!spdcc!dyer
From: geb@cs.pitt.edu (Gordon Banks)
Newsgroups: sci.med
Subject: Re: aids testing and insurance
Date: 17 Dec 92 15:58:11 GMT
Reply-To: geb@cs.pitt.edu (Gordon Banks)
Organization: Univ. of Pittsburgh Computer Science

In article <oneil.723943945@cwis> oneil@cwis.unomaha.edu (Sharon O'Neil) writes:
>Well, my recommendation is that if you plan to get tested for AIDS to 
>NOT do it at a blood bank.  Reason #1: If you get tested at a clinic, you 
>will get counseling.  Reason #2: If you test negative, it doesn't mean

You will receive counselling if you test positive for HIV at a blood
bank.

-- 
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Gordon Banks  N3JXP      | "I have given you an argument; I am not obliged
geb@cadre.dsl.pitt.edu   |  to supply you with an understanding." -S.Johnson
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Newsgroups: sci.med,sci.med.nutrition
From: dabbott@augean.eleceng.adelaide.edu.AU (Derek Abbott)
Subject: Vitamin C
Organization: Electrical and Electronic Eng., University of Adelaide
Date: Wed, 16 Dec 1992 07:33:44 GMT


I've found that megadosing myself with vit C can stop my cold
_if I get in there early_ and nip it in the bud.

However, once the cold sets in, I find that vit C actually prolongs
my cold. It seems better to let my body sweat it out unaided.

The vit C prolongs the cold, but keeps it on "medium." But without
the C, the cold gets more violent but geos away quicker.


Question: Has anyone else observed this or is it my imagination?

          If you reckon it's real, any theories as to why?
Newsgroups: sci.med
From: med50003@nusunix1.nus.sg (WANSAICHEONG KHIN-LIN)
Subject: Re: Skin Cancer
Organization: National University of Singapore
X-Newsreader: TIN [version 1.1 PL6]
Distribution: na
Date: Fri, 18 Dec 1992 17:47:41 GMT

Hmmm, the choice is the word 'skin cancer' is interesting. Be
definition, a cancer is malignant. A neoplasm may be benign or
malignant. If you are asking if there is some method of reversing the
damage that your mother has already been exposed to I don't think that
there are any established or widely accepted treatments.

The important things now are vigilant follow-up for the signs of early
malignancy and prevention of any further damage. If it isn't too
personal, what exactly was the histology of the lesions ?

gervais
From: pburch@roc.mbcr.bcm.tmc.edu (Paula Burch)
Newsgroups: sci.med
Subject: Re: female condom availability?
Date: 14 Dec 1992 17:58:59 GMT
Organization: Baylor College of Medicine, Houston, Tx
Distribution: na
Reply-To: pburch@bcm.tmc.edu (Paula Burch)
NNTP-Posting-Host: roc.mbcr.bcm.tmc.edu
Originator: pburch@roc.mbcr.bcm.tmc.edu


In article <1992Dec11.085752.21534@lmpsbbs.comm.mot.com>, markn@ssd.comm.mot.com (Mark Nowak) writes:
|> In article <71461@cup.portal.com> amigan@cup.portal.com (Mike - Medwid) writes:
|> >Only today did an FDA advisory panel give its qualified recommendation
|> >that the female condom be approved for sale in the US.  The company
|> >(Wisconsin something) claims a 12.5% failure (pregnancies in a 
|> >year) whle the panel felt it was likely higher.  The panel also felt
|> >it was not as effective as male condoms in the prevention of STDs.  
|> >The company claims a 5% failure rate if the device is used correctly.
|> 
|> I heard on the news today that the failure rate was as high as 25%.
|> They're trying to get the FDA to approve sales of it with a warning
|> label on it anyway.

The contraceptive sponge, according to a pamphlet I read, has failure rates 
up to 18%--about as good as withdrawal, which everybody agrees is not an 
adequate form of contraception. Several people on misc.kids have posted that 
they became pregnant while using the contraceptive sponge, although the 
mother was breastfeeding an infant (I almost said 'at the time', but that 
does draw up some odd pictures :-)), and breastfeeding does lower fertility dramatically.

Why aren't the ranges of failure rates routinely required to be listed on 
the outsides of the packages of all such prodcucts? They aren't even included 
in package inserts. 

Paula Burch
pburch@bcm.tmc.edu
Newsgroups: sci.med.aids
From: radiohc@ceniai.cu (Radio Habana Cuba)
Subject: Radio Havana Cuba Dec. 7, 1992
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Archive-Number: 6628
Organization: unspecified
Date: Mon, 14 Dec 92 04:04:32 PST

CUBA MARKS WORLD DAY AGAINST AIDS

Havana, Dec. 7(RHC)--  CONDI, a condom sporting a happy face and
a pair of big feet, walked along Havana's sea side drive Saturday
afternoon, inviting teenagers to think about safe sex.  CONDI is
the logo of of the UJC-- Union of Young Communists-- AIDS
prevention campaign.

Cuban rock stars Tanya and Edesio Alejandro joined voices
Saturday afternoon to wrap up activities marking World Day
against AIDS. Hundreds of Havana teens gathered at the city's
seaside drive to attend the launching of the AIDS education
campaign, promoted by the Union of Young Communists and the
National Center for Health Education.

Under the banner HAZLO CONMIGO--DO IT WITH ME--the campaign is
aimed to promote the use of condoms.  In Cuba, direct sexual
contact is the single most important primary source of AIDS
infection, since intravenous drug use is virtually nill; and all
blood supplies and donations are screened for the HIV-virus.
Maternal-fetal transmission is also practically non-existent, and
all expectant mothers receive comprehensive, free prenatal
checkups --including HIV tests.

Sixty percent of HIV-positive people in Cuba contracted the virus
through heterosexual intercourse; and nearly 40% through
homosexual intercourse.  In all cases, the average age of people
testing HIV-positive is going down:  the average age of Cuba's
over 800 seropositive persons has dipped to 19. In Cuba as a
whole, 54% of the population is under 30. "The alarming issue,"
says psychologist Lourdes Flores, "is that young people have a
sense of false security, because AIDS is not an epidemic in Cuba
and because public education has not reached them to convince
them they are in danger".

AIDS will affect some 20 million people worldwide by the year
2000, according to statistics released this summer at the 9th
International Conference on AIDS and Sexually Transmitted
Diseases, held in Holland.  In Cuba, 81 people in a population of
nearly 11 million have died from AIDS.  Some 12 million tests
have been carried out.

According to Psychologist Manuel Hernandez, the island carried
out a series of activities prior to  the World Day against AIDS,
December 1st.  Every province organized its own set of activities
ranging from bicycle competitions, live-music concerts and
theater. And El Pabellon Cuba, a popular teen center in the heart
of downtown Havana, aired documentaries on AIDS prevention,
followed by discussions led by health specialists.

During Saturday's activities, singer Hugo Lopez, one of the AIDS
patients at Havana's sanatorium, performed for the young crowd
and some 700 T-shirts with the banner of the campaign silk-
screened on them, were distributed among participants.

Newsgroups: sci.med.aids
From: Billi Goldberg <bigoldberg@igc.apc.org>
Subject: Vaginal Infection by HIV
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Archive-Number: 6635
Organization: unspecified
Date: Tue, 15 Dec 92 13:09:46 PST

Interesting new study that might help to explain the mucosal infection 
of HIV through vaginal and/or rectal exposure and the inability of HIV 
Ab to clear the infection..
------------------------------------------------------------------------

Hussain LA, Kelly CG, Fellowes R, Hecht EM, Wilson J, Chapman M, Lehner 
T. Expression and gene transcript of Fc receptors for IgG, HLA class II 
antingens and Langerhans cells in human cervico-vaginal epithelium. 
Clincal and Experimental Immunology (1992) 90, 530-538.

Abstract: The mechanism of transmission of HIV from the male to the 
female genital tract or in the reverse order is not clear. CD4 
glycoprotein is the receptor for HIV and Langerhans cells and the 
related dendritic cells could play a role in the initial transmission of 
HIV. Fc receptors (FcR) for IgG might be involved in antibody-mediated 
binding of HIV. We carried out an immunohistological study of normal 
human cervical and vaginal epithelia for the presence of CD4 
glycoprotein. Langerhans cells and FcR to IgG, CD4+ glycoprotein was not 
found in the vaginal or cervical epithelium, with the exception of a few 
endocervical epithelial cells. A small number of CD4+ mononuclear cells 
were found in the endocervical epithelium of a third of the specimens 
but a large number of CD4+ cells was found in the submucosa of most of 
the cervical and vaginal specimens. Langerhans cells expressing CD4, HLA 
class II, FcyR2 and FcyR3 were detected in most vaginal, ectocervical 
and transformation zone epithelia and in 9/14 endocervical issues. FcyR3 
was detected in about two-thirds of the columna endocervical epithelial 
cells and transformation zone by in situ hybridization, using a CD16-RNA 
probe. FcyR3 and FcyR2 gene transcripts were also found in fetal 
cervical tissue by applying the polymerase chain reaction to amplify 
portions of the FcyR3 and FcyR2 coding sequences in cDNA prepared from 
fetal RNA. HLA-DR was found in the endocervical cells, transformation 
zone and in Langerhans cells of all specimens. The presence of 
Langerhans cells, Fcy receptors and HLA class II antigen offers three 
potential mechanism for cervico-vaginal HIV transmission: (i) direct HIV 
infection of Langerhans cells, (ii) binding of HIV antibody complexes to 
cervical epithelial Fcy receptors and (iii) binding of HIV infected CD4+ 
cells to cervical HLA class II antigen which may infect these or the 
adjacent CD4+ cells.
Newsgroups: sci.med.aids
From: newell@netcom.com (newell neal)
Subject: AIDS INFO.
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Archive-Number: 6620
Organization: Netcom - Online Communication Services  (408 241-9760 guest) 
Date: Sat, 12 Dec 1992 06:37:51 GMT

 will try this again.
I am a Program Manager/HIV Team at a hospital in San Jose, CA. We
have staff and patients of many national groups and languages.
What I am looking for is information written in "proper Mexican/Spanish"
as opposed to "Chicano". Yes there is a deference. So what I need is AIDS.
information written for people who grew up in Mexico, not for people who
grew up in the US. If you have any sources please e-mail me.
Thank You
newell@netcom.com

Newsgroups: sci.med.aids
From: martin@sklib.usask.ca
Subject: ""Aids: What the Government isn't telling you""
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Archive-Number: 6641
Organization: University of Saskatchewan
Date: 16 DEC 92 16:07:24 GMT

I do not subscribe to this newsgroup, nor am I a regular reader, so please
excuse me if I do not use proper etiquette in sending this message.   I am
looking for information on a book written by Lorraine Day in 1991.  The title
is "Aids: What the government isn't telling you".  Has anyone seen any articles
or reviews, favourable or not, on this book?  I would greatly appreciate any
information that anyone out there has on this item. (The ISBN is 0-963-
0940-0-9, the LC card number is LC 91-75346, it was published in 1991 by
rockford ross.)
If possible, could responses be sent directly to me at "martin@sklib.usask.ca"

Many thanks in advance,

Colleen Martin
Reference Dep't
University of Saskatchewan
Newsgroups: sci.med.aids
From: larry@bradley.bradley.edu (L. D. Stratton)
Newsgroups: sci.med.aids
Subject: Re: ""Aids: What the Government isn't telling you""
Date: 16 Dec 92 19:06:41 GMT
Organization: Bradley University
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Archive-Number: 6645

In <1992Dec16.173307.8977@cs.ucla.edu> martin@sklib.usask.ca writes:
>I do not subscribe to this newsgroup, nor am I a regular reader, so please
>excuse me if I do not use proper etiquette in sending this message.   I am
>looking for information on a book written by Lorraine Day in 1991.  The title
>is "Aids: What the government isn't telling you".  Has anyone seen any articles
>information that anyone out there has on this item. (The ISBN is 0-963-
>0940-0-9, the LC card number is LC 91-75346, it was published in 1991 by
>rockford ross.)
>If possible, could responses be sent directly to me at "martin@sklib.usask.ca"

>Many thanks in advance,
>Colleen Martin
>Reference Dep't
>University of Saskatchewan

   Colleen,
       You should know that Dr. Lorraine Day is extremely homophobic and
       considered a fringe-extremist in her views of HIV transmission.
Newsgroups: sci.med.aids
From: Billi Goldberg <bigoldberg@igc.apc.org>
Subject: CMV and Lymphocytes
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Archive-Number: 6656
Organization: unspecified
Date: Fri, 18 Dec 92 21:40:46 PST

               CMV AND PERIPHERAL BLOOD LYMPHOCYTES

The study below is quite interesting. CMV is usually treated with 
ganciclovir and/or foscarnet both of which have potentially serious side 
effects including immunosuppression. Outbreaks of CMV have not been seen 
in individuals using DNCB. I have not mentioned this because there was 
nothing to support that DNCB could be effective against CMV. But, we 
know from the pilot study that the CD8+ subsets especially the CD8+ HLA-
DR and the CD56+ natural killer subset increase during DNCB usage.

This is not intended as any sort of claim for DNCB but only for immune 
modulation. Any type of treatment that can initiate a systemic boost to 
the immune system will result in increases in these subsets. CMV 
infections shows up only when AIDS progresses and the immune response is 
no longer able to control CMV.

Reprints are available from Dr. A.P. van den Berg, Department of 
Clinical Immunology, University Hospital, Oostersingel 59, 9713 EZ 
Groningen, Netherlands.
************************************************************************
van den Berg AP, van Son WJ, Janssen RAJ, Brons NHC, Heyn AA, Scholten-
Sampson A, Postma S, van der Giessen M, Tegzess AM, de Leij LHFM, The 
TH. Recover from Cytomegalovirus Infection is Associated with Activation 
of Peripheral Blood Lymphocytes December 1992;166:1228-35.

Abstract: The number of CD8(bright)* and CD56+ lymphocytes in the 
peripheral blood and their activation status were monitored by flow 
cytometry in 23 renal transplant recipients with cytomegalovirus (CMV) 
infection and were correlated with the virus load (as determined by CMV 
antigenemia) and clinical symptoms. Recovery from CMV infection 
coincided with expansion of the CD8(bright) and CD56+ subsets and with 
increased expression of the activation marker HLA-DR. Primary infection 
was association with activation of both subsets, whereas during 
secondary infection, mainly CD8(bright) cells responded. Progressive CMV 
disease (requiring antiviral treatment) and relapse occurred in 
association with low number of activated CD8(bright) and CD56+ cells. 
Lymphocyte activation and antibody responses against CMV often occurred 
simultaneously, but different kinetics of these responses in some 
patients indicated that cellular responses are necessary to control 
viral replication, whereas humoral responses alone may be insufficient. 
Monitoring of lymphocyte activation may provide clinically useful 
information during CMV infection.

*CD8(bright): The CD8 subset was divided into bright and dim fluorescent 
subpopulations by manual adjustment of the gate. Only the CD8(bright) cells 
(representing cytotoxic/suppressor cells) were enumerated.
Newsgroups: sci.med.aids
From: page@writer.yorku.ca (Page Westcott)
Subject: CMV
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Keywords: CMV, foscarnet, gancyclovir
Archive-Number: 6660
Organization: Computer Assisted Writing Centre, York University, Toronto, Canada
Date: 	Sun, 20 Dec 1992 14:12:45 -0500

A friend of mine who has been taking foscarnet for almost a year for
CMV Retinitis has recently been asked to take part in a study which
involves injecting gancyclovir directly into the eyeball and reducing
the amount of foscarnet taken daily.  I believe he is taking 350ml of
foscarnet daily, 5 days a week and will reduce this frequency to only
twice a week.  

Has anyone heard of this treatment and if so the success rate?  His
sight has not deteriorated very much in the past 12 months, and it
seems to me it would be better to leave well enough alone at this
point in his treatment.

Thanks, P.
Newsgroups: sci.med.aids
From: rad!arthur@uunet.UU.NET (Arthur Hu)
Subject: How to contact WHO
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Archive-Number: 6648
Organization: unspecified
Date: Wed, 16 Dec 92 13:45:50 PST

Where is the World Health Organization?

The CDC is easy to locate (it's in Atlanta), but I couldn't find
WHO except the U.N. said you've got to get them in Europe or
somewhere like that. 

Where can you get WHO statistics on AIDS for nations around the world?
Newsgroups: sci.med.aids
From: weemba@sagi.wistar.upenn.edu (Matthew P Wiener)
Subject: Re: Another Interesting Book
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Reply-To: weemba@sagi.wistar.upenn.edu (Matthew P Wiener)
Archive-Number: 6624
Organization: The Wistar Institute of Anatomy and Biology
Date: 13 Dec 92 17:51:42 GMT

In article <1992Dec12.181127.17806@cs.ucla.edu>, bigoldberg@igc (Billi Goldberg) writes:
>It provides another focus of AIDS contrary to the establishment. [...]
>It is hard for me to fathom how using drugs for the treatment of HIV 
>that are immunosuppressive can help an already suppressed immune system.

Many theories try to explain AIDS as an autoimmune disease.  So the goal
is to turn off the improper antiself reaction.

One version [1] posits the immune network being put off-kilter by gp120.
MHC(II) is one of the fundamentals of the immune network, and gp120 is
an `internal image' of it--CD4 binds to MHC(II), and gp120 binds to CD4.
So antibodies raised against gp120 also attack MHC(II), destroying the
network.

Another version [2] views gp120 as an MHC(II) mimic.  HIV sheds gp120,
and this continually stimulates CD4+ cells, much more than the normal
levels of MHC(II).  At some point, these cells stop responding.

Trials with cyclosporin have been done.  It seems to be helpful at early
stages [3], but murderous at advanced stages [4].  The latter trials,
plus the "common knowledge" that AIDS _is_ immunosuppression, seems to
have dampened any interest in further such trials.

[1] G W Hoffmann, T A Kion, M D Grant "An idiotypic network model of
    AIDS immunopathenogenesis" PNAS 88:3060 (1991).

[2] M S Ascher, H W Sheppard "AIDS an Immune System Activation. II.
    The Panergic Imnesia Hypothesis" J AIDS 3:177 (1990).

[3] J-M Andrieu et al "Effects of cyclosporin on T-cell subsets in
    human immunodeficiency virus disease" CLIN IMMUNOL IMMUNOPATH
    46:181 (1988).

[4] A Phillips et al "Cyclosporine-induced deterioration in patients
    with AIDS" CMAJ 140:1456 (1989).
-- 
-Matthew P Wiener (weemba@sagi.wistar.upenn.edu)
From: hank@netcom.com (Hank Roberts)
Subject: Re: ""Aids: What the Government isn't telling you""
Note: Copyright 1992, Dan R. Greening. Non-commercial reproduction allowed.
Archive-Number: 6647
Organization: Netcom - Online Communication Services  (408 241-9760 guest) 
Date: Thu, 17 Dec 1992 01:33:36 GMT

Try the magazine and newspaper indexes; Dr. Day is or was a surgeon at
Stanford; I recall one interview in which she espressed concerna bout
breathing the 'mist of blood and bone" that results from using power saws
to open the ribcage during major surgery.  I think she was an early user
of full respirator gear as a surgeon.
Newsgroups: sci.med.aids