Before revision:  This behaviour is predicted by a model that assumes that deaths are caused by the degradation of the ACE2 receptor by the virus , which uses it as entry point in the cells. The insufficient levelof ACE2 exacerbates the inflammatory response eventually leading to death, but if the initial levels of ACE2 are high the immune system has enough time to resolve the infection before it gets to extreme consequences.
After revision:  This behaviour is predicted by a model that assumes that deaths are caused by the degradation of the ACE2 and thus worsens lung injury, causes vasoconstriction, possible thrombotic problems and exacerbates the inflammatory response eventually leading to death, but if the initial levels of ACE2 are high the immune system has enough time to resolve the infection before it gets to extreme consequences.