Reabsorption - Wikipedia Reabsorption Not to be confused with Resorption. This article has multiple issues. Please help improve it or discuss these issues on the talk page. ( Learn how and when to remove these template messages ) This article does not cite any sources. Please help improve this article by adding citations to reliable sources. Unsourced material may be challenged and removed. ( December 2009 ) This article may be confusing or unclear to readers. Please help us clarify the article. There might be a discussion about this on the talk page. ( February 2007 ) In renal physiology, reabsorption or tubular reabsorption is the process by which the nephron removes water and solutes from the tubular fluid ( pre-urine ) and returns them to the circulating blood. It is called reabsorption ( and not absorption ) both because these substances have already been absorbed once ( particularly in the intestines ) and because the body is reclaiming them from a post glomerular fluid stream that is well on its way to becoming urine ( that is, they will soon be lost to the urine unless they are reclaimed ). Substances are reabsorbed from the tubule into the peritubular capillaries. This happens as a result of sodium transport from the lumen into the blood by the Na / K ATPase in the basolateral membrane of the epithelial cells. Thus, the glomerular filtrate becomes more concentrated, which is one of the steps in forming urine. Reabsorption allows many useful solutes ( primarily glucose and amino acids ), salts and water that have passed through Bowman 's capsule, to return to the circulation. These solutes are reabsorbed isotonically, in that the osmotic potential of the fluid leaving the proximal convoluted tubule is the same as that of the initial glomerular filtrate. However, glucose, amino acids, inorganic phosphate, and some other solutes are reabsorbed via secondary active transport through cotransport channels driven by the sodium gradient. Renin -- angiotensin system : 1. The kidneys sense low blood pressure. 2. Release renin into the blood. 3. Renin causes production of Angiotensin I. 4. Angiotensin Converting Enzyme ( ACE ) converts Angiotensin I to Angiotensin II. 5. Angiotensin II stimulates the release of Aldosterone, ADH, and thirst. 6. Aldosterone causes kidneys to reabsorb sodium ; ADH increases the uptake of water. 7. Water follows sodium. 8. As blood volume increases, pressure also increases. See also Transepithelial potential difference driving reabsorption Retrieved from `` https://en.wikipedia.org/w/index.php?title=Reabsorption&oldid=770123158 '' Categories : Renal physiology Hidden categories : Articles lacking sources from December 2009 All articles lacking sources Wikipedia articles needing clarification from February 2007 All Wikipedia articles needing clarification Talk About Wikipedia Русский Edit links This page was last edited on 13 March 2017, at 15 : 27. Text is available under the Creative Commons Attribution - ShareAlike License ; additional terms may apply. By using this site, you agree to the Terms of Use and Privacy Policy. Wikipedia ® is a registered trademark of the Wikimedia Foundation, Inc., a non-profit organization. About Wikipedia

Question: where are amino acids reabsorbed in the nephron
Answer: from the tubule into the peritubular capillaries