Heart sounds - wikipedia Heart sounds Normal heart sounds Normal heart sounds as heard with a stethoscope Problems playing this file ? See media help. 72 BPM Heart sound Front of thorax, showing surface relations of bones, lungs ( purple ), pleura ( blue ), and heart ( red outline ). The locations of best auscultation for each heart valve are labeled with `` M '', `` T '', `` A '', and `` P ''. First heart sound : caused by atrioventricular valves - Mitral ( M ) and Tricuspid ( T ). Second heart sound caused by semilunar valves -- Aortic ( A ) and Pulmonary / Pulmonic ( P ). Diagram showing relations of opened heart to front of thoracic wall. Ant. Anterior segment of tricuspid valve. A O. Aorta. A.P. Anterior papillary muscle. In. Innominate artery. L.C.C. Left common carotid artery. L.S. Left subclavian artery. L.V. Left ventricle. P.A. Pulmonary artery. R.A. Right atrium. R.V. Right ventricle. V.S. Ventricular septum. Heart sounds are the noises generated by the beating heart and the resultant flow of blood through it. Specifically, the sounds reflect the turbulence created when the heart valves snap shut. In cardiac auscultation, an examiner may use a stethoscope to listen for these unique and distinct sounds that provide important auditory data regarding the condition of the heart. In healthy adults, there are two normal heart sounds often described as a lub and a dub ( or dup ), that occur in sequence with each heartbeat. These are the first heart sound ( S ) and second heart sound ( S ), produced by the closing of the atrioventricular valves and semilunar valves, respectively. In addition to these normal sounds, a variety of other sounds may be present including heart murmurs, adventitious sounds, and gallop rhythms S and S. Heart murmurs are generated by turbulent flow of blood, which may occur inside or outside the heart. Murmurs may be physiological ( benign ) or pathological ( abnormal ). Abnormal murmurs can be caused by stenosis restricting the opening of a heart valve, resulting in turbulence as blood flows through it. Abnormal murmurs may also occur with valvular insufficiency ( regurgitation ), which allows backflow of blood when the incompetent valve closes with only partial effectiveness. Different murmurs are audible in different parts of the cardiac cycle, depending on the cause of the murmur. Contents 1 Primary heart sounds 2 Extra heart sounds 3 Murmurs 3.1 Effects of breathing 3.2 Interventions that change murmurs 4 Other abnormal sounds 5 Surface anatomy 6 Recording heart sounds 7 See also 8 Notes and references 9 External links Primary heart sounds Normal heart sounds are associated with heart valves closing : S The first heart sound, or S, forms the `` lub '' of `` lub - dub '' and is composed of components M ( mitral valve closure ) and T ( tricuspid valve closure ). Normally M precedes T slightly. It is caused by the closure of the atrioventricular valves, i.e. tricuspid and mitral ( bicuspid ), at the beginning of ventricular contraction, or systole. When the ventricles begin to contract, so do the papillary muscles in each ventricle. The papillary muscles are attached to the cusps or leaflets of the tricuspid and mitral valves via chordae tendineae ( heart strings ). When the papillary muscles contract, the chordae tendineae become tense and thereby prevent the backflow of blood into the lower pressure environment of the atria. The chordae tendineae act a bit like the strings on a parachute, and allow the leaflets of the valve to balloon up into the atria slightly, but not so much as to evert the cusp edges and allow back flow of blood. It is the pressure created from ventricular contraction that closes the valve, not the papillary muscles themselves. The contraction of the ventricle begins just prior to AV valves closing and prior to the semilunar valves opening. The sudden tensing of the chordae tendineae and the squeezing of the ventricles against closed semilunar valves, sends blood rushing back toward the atria, and the parachute - like valves catch the rush of blood in their leaflets causing the valve to snap shut. The S1 sound results from reverberation within the blood associated with the sudden block of flow reversal by the valves. The delay of T1 even more than normally causes the split S1 which is heard in a right bundle branch blockage. The second heart sound, or S, forms the `` dub '' of `` lub - dub '' and is composed of components A ( aortic valve closure ) and P ( pulmonary valve closure ). Normally A precedes P especially during inhalation where a split of S can be heard. It is caused by the closure of the semilunar valves ( the aortic valve and pulmonary valve ) at the end of ventricular systole and the beginning of ventricular diastole. As the left ventricle empties, its pressure falls below the pressure in the aorta. Aortic blood flow quickly reverses back toward the left ventricle, catching the pocket - like cusps of the aortic valve, and is stopped by aortic valve closure. Similarly, as the pressure in the right ventricle falls below the pressure in the pulmonary artery, the pulmonary valve closes. The S sound results from reverberation within the blood associated with the sudden block of flow reversal. Splitting of S2, also known as physiological split, normally occurs during inhalation because the decrease in intrathoracic pressure increases the time needed for pulmonary pressure to exceed that of the right ventricular pressure. A widely split S2 can be associated with several different cardiovascular conditions, including left bundle branch block, pulmonary stenosis, and atrial septal defect. Extra heart sounds The rarer extra heart sounds form gallop rhythms and are heard in both normal and abnormal situations. Main article : Third heart sound Rarely, there may be a third heart sound also called a protodiastolic gallop, ventricular gallop, or informally the `` Kentucky '' gallop as an onomatopoeic reference to the rhythm and stress of S1 followed by S2 and S3 together ( S1 = Ken ; S2 = tuck ; S3 = y ). `` lub - dub - ta '' or `` slosh - ing - in '' If new, indicates heart failure or volume overload. It occurs at the beginning of diastole after S2 and is lower in pitch than S1 or S2 as it is not of valvular origin. The third heart sound is benign in youth, some trained athletes, and sometimes in pregnancy but if it re-emerges later in life it may signal cardiac problems, such as a failing left ventricle as in dilated congestive heart failure ( CHF ). S3 is thought to be caused by the oscillation of blood back and forth between the walls of the ventricles initiated by blood rushing in from the atria. The reason the third heart sound does not occur until the middle third of diastole is probably that during the early part of diastole, the ventricles are not filled sufficiently to create enough tension for reverberation. It may also be a result of tensing of the chordae tendineae during rapid filling and expansion of the ventricle. In other words, an S3 heart sound indicates increased volume of blood within the ventricle. An S3 heart sound is best heard with the bell - side of the stethoscope ( used for lower frequency sounds ). A left - sided S3 is best heard in the left lateral decubitus position and at the apex of the heart, which is normally located in the 5th left intercostal space at the midclavicular line. A right - sided S3 is best heard at the lower - left sternal border. The way to distinguish between a left and right - sided S3 is to observe whether it increases in intensity with inhalation or exhalation. A right - sided S3 will increase on inhalation, while a left - sided S3 will increase on exhalation. S3 can be a normal finding in young patients but is generally pathologic over the age of 40. The most common cause of pathologic S3 is congestive heart failure. Main article : Fourth heart sound S4 when audible in an adult is called a presystolic gallop or atrial gallop. This gallop is produced by the sound of blood being forced into a stiff or hypertrophic ventricle. `` ta - lub - dub '' or `` a-stiff - wall '' It is a sign of a pathologic state, usually a failing or hypertrophic left ventricle, as in systemic hypertension, severe valvular aortic stenosis, and hypertrophic cardiomyopathy. The sound occurs just after atrial contraction at the end of diastole and immediately before S1, producing a rhythm sometimes referred to as the `` Tennessee '' gallop where S4 represents the `` Ten - '' syllable. It is best heard at the cardiac apex with the patient in the left lateral decubitus position and holding his breath. The combined presence of S3 and S4 is a quadruple gallop, also known as the `` Hello - Goodbye '' gallop. At rapid heart rates, S3 and S4 may merge to produce a summation gallop, sometimes referred to as S7. Atrial contraction must be present for production of an S4. It is absent in atrial fibrillation and in other rhythms in which atrial contraction does not precede ventricular contraction. Murmurs Auscultogram from normal and abnormal heart sounds. Heart murmurs are produced as a result of turbulent flow of blood strong enough to produce audible noise. They are usually heard as a whooshing sound. The term murmur only refers to a sound believed to originate within blood flow through or near the heart ; rapid blood velocity is necessary to produce a murmur. It should be noted that most heart problems do not produce any murmur and most valve problems also do not produce an audible murmur. Murmurs can be heard in many situations in adults without major congenital heart abnormalities : Regurgitation through the mitral valve is by far the most commonly heard murmur, producing a pansystolic / holosystolic murmur which is sometimes fairly loud to a practiced ear, even though the volume of regurgitant blood flow may be quite small. Yet, though obvious using echocardiography visualization, probably about 20 % of cases of mitral regurgitation do not produce an audible murmur. Stenosis of the aortic valve is typically the next most common heart murmur, a systolic ejection murmur. This is more common in older adults or in those individuals having a two, not a three leaflet aortic valve. Regurgitation through the aortic valve, if marked, is sometimes audible to a practiced ear with a high quality, especially electronically amplified, stethoscope. Generally, this is a very rarely heard murmur, even though aortic valve regurgitation is not so rare. Aortic regurgitation, though obvious using echocardiography visualization, usually does not produce an audible murmur. Stenosis of the mitral valve, if severe, also rarely produces an audible, low frequency soft rumbling murmur, best recognized by a practiced ear using a high quality, especially electronically amplified, stethoscope. Other audible murmurs are associated with abnormal openings between the left ventricle and right heart or from the aortic or pulmonary arteries back into a lower pressure heart chamber. Gradations of Murmurs ( Defined based on use of an acoustic, not a high - fidelity amplified electronic stethoscope ) Grade Description Grade 1 Very faint, heard only after listener has `` tuned in '' ; may not be heard in all positions. Only heard if the patient `` bears down '' or performs the Valsalva maneuver. Grade 2 Quiet, but heard immediately after placing the stethoscope on the chest. Grade 3 Moderately loud. Grade 4 Loud, with palpable thrill ( a tremor or vibration felt on palpation ) Grade 5 Very loud, with thrill. May be heard when stethoscope is partly off the chest. Grade 6 Very loud, with thrill. May be heard with stethoscope entirely off the chest. Though several different cardiac conditions can cause heart murmurs, the murmurs can change markedly with the severity of the cardiac disease. An astute physician can sometimes diagnose cardiac conditions with some accuracy based largely on the murmur, related physical examination, and experience with the relative frequency of different heart conditions. However, with the advent of better quality and wider availability of echocardiography and other techniques, heart status can be recognized and quantified much more accurately than formerly possible with only a stethoscope, examination, and experience. Another advantage to the use of the echocardiogram is that the devices can be hand held. Effects of breathing Inhalation pressure causes an increase in the venous blood return to the right side of the heart by decreasing intrathoracic pressure making it more negative ( pulling blood into the right side of the heart via a vacuum - like effect ). Therefore, right - sided murmurs generally increase in intensity with inhalation. The decreased ( more negative ) intrathoracic pressure has an opposite effect on the left side of the heart, making it harder for the blood to exit into circulation. Therefore, left - sided murmurs generally decrease in intensity during inhalation. Inhalation can also produce a non-pathological split S2 which will be heard upon auscultation. With exhalation, the opposite haemodynamic changes occur : left - sided murmurs generally increase in intensity with exhalation. If a patient lies supine with his legs up at a 45 - degree angle, venous return to the right side of the heart produces effects similar to inhalation - increased blood flow. Interventions that change murmurs Main article : Heart murmur There are a number of interventions that can be performed that alter the intensity and characteristics of abnormal heart sounds. These interventions can differentiate the different heart sounds to more effectively obtain a diagnosis of the cardiac anomaly that causes the heart sound. Other abnormal sounds Clicks -- Heart clicks are short, high - pitched sounds that can be appreciated with modern non-invasive imaging techniques. Rubs -- The pericardial friction rub can be heard in pericarditis, an inflammation of the pericardium, the sac surrounding the heart. This is a characteristic scratching, creaking, high - pitched sound emanating from the rubbing of both layers of inflamed pericardium. It is the loudest in systole, but can often be heard at the beginning and at the end of diastole. It is very dependent on body position and breathing, and changes from hour to hour. Surface anatomy The aortic area, pulmonic area, tricuspid area and mitral area are areas on the surface of the chest where the heart is auscultated. Heart sounds result from reverberation within the blood associated with the sudden block of flow reversal by the valves closing. Because of this, auscultation to determine function of a valve is usually not performed at the position of the valve, but at the position to where the sound waves reverberate. Aortic valve ( to aorta ) right second intercostal space upper right sternal border Pulmonary valve ( to pulmonary trunk ) left second intercostal space upper left sternal border Erb 's point Left third intercostal space left sternal border Tricuspid valve ( to right ventricle ) left fourth, fifth intercostal spaces lower left sternal border Mitral valve ( to left ventricle ) left fifth intercostal space left midclavicular line Recording heart sounds Using electronic stethoscopes, it is possible to record heart sounds via direct output to an external recording device, such as a laptop or MP3 recorder. The same connection can be used to listen to the previously recorded auscultation through the stethoscope headphones, allowing for more detailed study of murmurs and other heart sounds, for general research as well as evaluation of a particular patient 's condition. See also Pulse Physical examination Precordial examination Benign pediatric heart murmur Iambic pentameter, a metre in poetry which follows a similar rhythm to the human heartbeat Pulsatile tinnitus - hearing a heartbeat sound in one or both ears Notes and references Jump up ^ https://depts.washington.edu/physdx/heart/tech2.html Jump up ^ https://depts.washington.edu/physdx/heart/tech2.html Jump up ^ `` thrill ''. Medline Plus Medical Dictionary. Jump up ^ Bernstein, Lenny ( 2016 - 01 - 02 ), `` Heart doctors are listening for clues to the future of their stethoscopes '', Washington Post. External links University of Michigan Heart Sound and Murmur Library. Auscultation Assistant. UCLA Heart Sounds - Heart Murmurs. practicalclinicalskills.com Physiology of the cardiovascular system Heart Cardiac output Cardiac cycle Cardiac output Heart rate Stroke volume Stroke volume End - diastolic volume End - systolic volume Afterload Preload Frank -- Starling law Cardiac function curve Venous return curve Wiggers diagram Pressure volume diagram Ultrasound Fractional shortening = ( End - diastolic dimension End - systolic dimension ) / End - diastolic dimension Aortic valve area calculation Ejection fraction Cardiac index Heart rate Cardiac pacemaker Chronotropic ( Heart rate ) Dromotropic ( Conduction velocity ) Inotropic ( Contractility ) Bathmotropic ( Excitability ) Lusitropic ( Relaxation ) Conduction Conduction system Cardiac electrophysiology Action potential cardiac atrial ventricular Effective refractory period Pacemaker potential Electrocardiography P wave PR interval QRS complex QT interval ST segment T wave U wave Hexaxial reference system Chamber pressure Central venous Right atrial ventricular pulmonary artery wedge Left atrial ventricular Aortic Other Ventricular remodeling Vascular system / Hemodynamics Blood flow Compliance Vascular resistance Pulse Perfusion Blood pressure Pulse pressure Systolic Diastolic Mean arterial pressure Jugular venous pressure Portal venous pressure Regulation of BP Baroreflex Kinin -- kallikrein system Renin -- angiotensin system Vasoconstrictors Vasodilators Autoregulation Myogenic mechanism Tubuloglomerular feedback Cerebral autoregulation Paraganglia Aortic body Carotid body Glomus cell Medical examination and history taking Medical history Chief complaint History of the present illness Systems review Nursing assessment Allergies Medications Past medical history Surgical history Family history Social history Psychiatric history Progress notes Mnemonics SAMPLE OPQRST SOAP Physical examination General / IPPA Inspection Auscultation Palpation Percussion Vital signs Temperature Heart rate Blood pressure Respiratory rate HEENT Oral mucosa TM Eyes ( Ophthalmoscopy, Swinging - flashlight test ) Hearing ( Weber, Rinne ) Respiratory Respiratory sounds Cyanosis Clubbing Cardiovascular Precordial examination Peripheral vascular examination Heart sounds Other Jugular venous pressure Abdominojugular test Carotid bruit Ankle - brachial pressure index Abdominal Digestive Liver span Rectal Murphy 's sign Bowel sounds Urinary Murphy 's punch sign Extremities / Joint Back ( Straight leg raise ) Knee ( McMurray test ) Hip Wrist ( Tinel sign, Phalen maneuver ) Shoulder ( Adson 's sign ) GALS screen Neurological Mental state Mini -- mental state examination Cranial nerve examination Upper limb neurological examination Neonatal Apgar score Ballard Maturational Assessment Gynecological Well - woman examination Vaginal examination Breast examination Cervical motion tenderness Assessment and plan Medical diagnosis Differential diagnosis Symptoms and signs relating to the cardiovascular system ( R00 -- R03, 785 ) Chest pain Referred pain Angina Aerophagia Auscultation Heart sounds Split S2 S3 S4 Gallop rhythm Heart murmur Systolic Diastolic Continuous Pericardial friction rub Heart click Bruit carotid Pulse Tachycardia Bradycardia Pulsus tardus et parvus Pulsus paradoxus doubled Pulsus bisferiens Dicrotic pulse Pulsus bigeminus Pulsus alternans Pulse deficit Vascular disease Gangrene Other Palpitations Apex beat Cœur en sabot Jugular venous pressure Cannon A waves Hyperaemia Shock Cardiogenic Hypovolemic Distributive Septic Neurogenic Retrieved from `` https://en.wikipedia.org/w/index.php?title=Heart_sounds&oldid=798915355 '' Categories : Cardiology Audible medical signs Symptoms and signs : Cardiac Hidden categories : Articles with hAudio microformats Talk Contents About Wikipedia Български Català Čeština Deutsch Eesti Español Euskara فارسی Français 한국어 Hrvatski Bahasa Indonesia Italiano עברית Basa Jawa Nederlands Norsk nynorsk Polski Português Svenska ไทย 中文 Edit links This page was last edited on 4 September 2017, at 15 : 18. 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Question: what is the lub sound of the heart
Answer: first heart sound (S1)