Regardless of mechanism, IL-15-induced trafficking of lymphocytes is an important part of our overall understanding of influenza immunobiology. The studies presented here emphasize the importance of IL-15 in mediating the innate response to influenza via the trafficking of NK cells, as viral titers were not as efficiently controlled early after infection in IL-15-blocked animals. These and previous studies also suggest that exogenous IL-15c could be used to modulate the immune response at both the innate and adaptive phases. Overall, we believe that IL-15 may be in important point of eventual immune intervention for treatment of primary infection and for adjuvanting vaccines. Enterovirus 104 Infection in Adult, Japan, 2011   Screening for Influenza A(H1N1)pdm09, Auckland International Airport, New Zealand Entry screening for influenza A(H1N1)pdm09 at Auckland International Airport, New Zealand, detected 4 cases, which were later confirmed, among 456,518 passengers arriving April 27–June 22, 2009. On the basis of national influenza surveillance data, which suggest that ≈69 infected travelers passed through the airport, sensitivity for screening was only 5.8%. T he virus that caused the 2009 infl uenza pandemic, hereafter referred to as infl uenza A(H1N1)pdm09, is mainly spread internationally by air travel (1) . To prevent or delay such spread, during the pandemic many countries initiated screening of air travelers arriving at airports, even though these measures have not been recommended by the World Health Organization (2) . On April 25, 2009, New Zealand was one of the fi rst countries outside the Americas to confi rm infl uenza A(H1N1)pdm09 in arriving airline passengers (3) . During April 27-June 22, 2009, at the direction of the Ministry of Health, the Auckland Regional Public Health Service began a screening program at Auckland International Airport.