As mentioned above, both noroviruses and SaVs generally cause mild to asymptomatic enteric infections in human and animal hosts [12] . Human SaV infections are reported to be similar to or milder than human norovirus infections, but SaV infections have a shorter duration of viral shedding and are less associated with projectile vomiting [13] [14] [15] [16] . Incidence of SaV-associated gastroenteritis infections remains less than norovirusassociated infections for both sporadic and outbreak settings, though various studies have reported increasing rates of SaV infections around the world [17] [18] [19] [20] [21] . The genetic diversity of SaVs is comparable to that of noroviruses, and the diversity of reported animal hosts is also similar. Noroviruses have been discovered in specimens from humans, pigs, cattle, dogs, sea lions, African lion, and mice [22] [23] [24] [25] . In comparison, SaVs have been found in specimens from humans, pigs, dogs, minks and California sea lions [23, [25] [26] [27] .