All porcine NoVs were detected from pigs without clinical signs (9, 10) . Subclinically infected pigs may be natural reservoirs for NoVs, and because porcine GII NoVs are genetically and antigenically related to human NoVs, concerns exist about their zoonotic potential. Whether human NoV strains similar to the QW101-like porcine NoVs circulate among people with occupational exposure to pigs is unknown, but such studies could provide information on the zoonotic potential of these porcine NoVs. The RdRp-capsid junction region of NoVs contains a highly conserved 18-nt motif in genomic and subgenomic RNA that is believed to be a transcription start signal (1, 20) . All 18 nt were identical within each genogroup except for the Hu/GII/J23, Po/GII/QW101, and Po/GII/QW125 strains ( Figure 3B , sequence alignments on other GI and GIII strains are not shown). This finding suggests that homologous recombination may occur within this motif between NoVs of different genotypes within the same genogroup. Recombinant human GII NoVs have been reported previously (20) (21) (22) (23) (24) . To our knowledge, this study is the first identification of a potential recombinant between pig NoVs. At present, NoV recombinants have been detected exclusively between viruses within the same genogroup and within the same host species, but few animal NoVs have been sequenced (RdRp and capsid) for comparative analysis, especially those from animals in developing countries, where humans and animals may be in close contact.