Several hepatitis viruses such as Hepatitis A [20] , B [21, 22] , C [23] , and E, G [24] have been anticipated to be associated with this set of symptoms [7] . No association has been found with blood transfusion, toxins and drugs [17] . In one study Safadi and co-workers (2001) found out that sera of eight of the patients had the existence of Hepatitis Bc IgG and/or anti-HBs antibodies which was suggested due to past exposure and immunizing effect and they were unable to establish any direct relation with acute hepatitis B [14] . As non-A, non-B hepatitis agents are usually responsible for the hepatitis associated aplastic anemia; the prevalence of anti-hepatitis C virus antibodies is similar in HAAA and aplasia of other origins [25] . HCV seropositivity has been observed in the patients developing cytopenia following non-A non-B hepatitis (NANBH). HCV viremia has been frequently observed without detecting anti-HCV antibodies in patients' blood reflecting the transfusion associated HCV infection [25, 26] . However, it has also been reported that HCV is not generally implicated as a causative agent of hepatitis preceding aplastic anemia [27] .