The majority of microarray papers published with regard to epileptogenesis involve chemical kindling. These differ in terms of Tang) , directly compared the genomic expression changes with certain physiological and disease states. In this manner, several assertions were made. Elliott et al suggested support for the hypotheses of parallels existing between gene expression underlying development and epileptogenic plasticity during the latency period following comparisons with the adult and developing rats (4, 24, 95) .