Recent studies have shown that gelatinase B also known as matrix metalloproteinase-9 (MMP-9), an endopeptidase capable of degrading the extracellular matrix, is thought to be associated with atherosclerosis, and plaque rupture [12, 13] . Therefore, MMP-9 is considered to be an important mediator of vascular remodeling and plaque instability. The MMP-9 action is enhanced b neutrophil gelatinase-associated lipocalin (NGAL), also known as lipocalin-2, a 25 kDa glycoprotein, that is, found in the granules of human neutrophils, with many diverse functions, such as scavenger of bacterial products, modulator of inflammation, iron trafficking, and apoptosis [14] .