The amyloid cascade hypothesis for Alzheimer's disease: an appraisal for the development of therapeutics
Amyloid cascade hypothesis: Pathogenesis and therapeutic strategies in Alzheimer's disease
P3-228 Chip and HSP70 regulate tau ubiquitination, degradation and aggregation
Alzheimer disease therapy—moving from amyloid-β to tau
The familial Alzheimer's disease APPV717I mutation alters APP processing and Tau expression in iPSC-derived neurons.
Extreme cerebrospinal fluid amyloid beta levels identify family with late-onset Alzheimer's disease presenilin 1 mutation.