The Theory of Evolution is a lie. We did not come from monkeys.

Ok, I'm going to get to the evidence as quick as I can, but first I would like to quickly clear up some misconceptions about evolution that my opponent has put forth. 1)Evolution does not say we came from monkeys, it says we share a common ancestor with them 2)Mutations are usually neutral because most of our DNA is junk, and mutations are not induced by the change in environment, they are random. 3)Evolution does not say we came here by "chance," although the genetic mutations seem random, the natural selection is definitely not. We are anything but the product of chance. 4) Evolution is not contrary to God, don't let anyone tell you otherwise. Now that that's out of the way, let's get to the evidence. Endogenous Retrovirus (or ERVs): A Retrovirus stores its genetic information in RNA. (Not DNA). When a Retrovirus infects a cell, it converts its RNA genome into DNA, through a process known as reverse transcription. The DNA then inserts randomly into one of the host's chromosomes. If the virus infects one of your germs cells that make your gametes (your egg or sperm cells). And then if you have sex and reproduce after your germ cells that make your sex cells get infected, the viral DNA will be passed on to the next generation, and all subsequent generations. They will have the SAME ERV sequence at the same EXACT location in the entire genome (~3 billion base pairs) We know the ERV sequences come from retroviruses for two reasons 1)ERV sequences look exactly like the genome of retroviruses. 2)We can actually see ERVs form after a retrovirus infects a cell. It is also important to note that ERVs are mutated and rendered non-functional, and serves as a marker only. It is common knowledge that the human genome is quite similar to the chimpanzee genome. However upon further and closer examination we see certain "interesting" things. On the short arm of chromosome 10 we find a relic of a viral infection, the ERV sequence. On the exact same location in the chimp's DNA there is also an ERV. The chances of 1 pair of viruses inserting at the exact same location in humans and chimpanzees (without common descent) is about 1 in 3,000,000,000. Also, over on the short arm of Chromosome 1, there is a remnant of another viral infection. And exactly in the same location we find an ERV in the Chimpanzee DNA. The odds of two pairs of viruses inserting at perfectly matches locations without common descent is 1 in 4,500,000,000,000,000,000 Also, if we take a look on the long arm of chromosome 19, we find a third ERV. Guess what? There is a matching ERV in the exact same location in the chimp genome. The odds of us sharing THREE pairs of retroviruses without common descent are 1 in 4,500,000,000,000,000,000,000,000,000. Also, if we take a look on the short arm of chromosome 6, And another ERV at the exact same location in the chimp DNA. The odds of FOUR pairs of viruses inserting at perfectly matches locations without common descent are 1 in 3,375,000,000,000,000,000,000,000,000,000,000,000. Guess what? There's more. One the long arm of the X chromosome we again find an ERV with another exact match in the same exact location in chimp DNA. The odds of FIVE pairs of viruses inserting at perfectly matched locations without evolution being true is 1 in 2,025,000,000,000,000,000,000,000,000000,000,000,000,000,000. The chance that evolution is false just based on these 5 is equivalent to you and your friend randomly picking the same single molecule of water from all the world's oceans. And yet there is more still! There are SIXTEEN instances of human endogenous retroviruses having exact matches with chimp ERVs. The odds of SIXTEEN pairs of viruses inserting at perfectly matches locations, with evolution being false are LESS than 1 in 2,057,400,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000,000. AND THOSE ARE JUST THE K CLASS RETROVIRUSES! Our genome contains roughly 98,000 ERVs. And most appear in the same location in chimps, gorillas, orangutans, and macaques. The odds that we did not share a common ancestor with the other primates are so small that the chances are just not fathomable. My opponent asks for just one beneficial mutation, here are some mutations my opponent would consider beneficial: 1)There's a family in Germany who were already unusually strong. But in one case, one of their children was born with a double copy of an anti-myostatin mutation carried by both parents. The result is a Herculean kiddo who was examined at only a few days old for his unusually well-developed muscles. By four years old, he had twice the muscle mass of normal children, and half the fat. Pharmaceutical synthesis of this mutation is being examined for potential use against muscular dystrophy or sarcopenia. 2)And then there's a family in Connecticut who've been identified as having hyper dense, virtually unbreakable bones. A team of doctors at Yale traced the mutation to a gene that was the subject of an earlier study. In that study researchers showed that low bone density could be caused by a mutation that disrupts the function of a gene called LRP5. This clued them that a different mutation increased LRP5 function, leading to an opposite phenotype, that is, high bone density. According to their investigators, members of this family have bones so strong they rival those of a character in the Bruce Willis movie, 'Unbreakable'. 3) Kinfolk in the village of Limone Sul Garda in northern Italy have a mutation which gives them better tolerance of HDL serum cholesterol. Consequently this family has no history of heart attacks despite their high-risk dietary habits. This mutation was traced to a single common ancestor living in the 1700's, but has now spread to dozens of descendants. Genetic samples from this family are now being tested for potential treatment of patients of heart disease. 4) Another example of that is the CCR5-delta 32 mutation. About 10% of whites of European origin now carry it. But the incidence is only 2% in central Asia, and is completely absent among East Asians, Africans, and tribal Americans. It appears to have suddenly become relatively common among white Europeans about 700 years ago, evidently as a result of the Black Plague, indicating another example of natural selection allowing one gene dominance in a changing environment. It is harmless or neutral in every respect other than its one clearly beneficial feature. According to Science-Frontiers.com, if one inherits this gene from both parents, they will be especially resistant, if not immune to AIDS. How do I know the human race evolved? Simple! The human race is evolving RIGHT NOW! My opponent asks me how I know the human eye evolved. The answer is simple: I know the human eye evolved because the HUMAN EYE US EVOLVING RIGHT NOW! We've identified an emerging population of tetra chromatic women who can see a bit of the normally invisible ultraviolet spectrum. Normally humans are tri chromatic with 3 cones, but women who are carriers for variant cone pigments might be born as full tetrachromats, having four different simultaneously functioning kinds of cones to pick up different colors. We can also show which animals share common ancestors within different species due to their shared atavisms. An Atavism is the tendency to revert to ancestral type. An atavism is an evolutionary throwback, such as traits reappearing which had disappeared generations ago. This explains how birds still have genes for making teeth, humans still have genes for making tails, and whales still have the genes for making legs. This also explains how some women can be borne (rare case) with extra nipples. The extra nipples follow down the milk line. (Think of a pig or dog and how their nipples are set up.) (I'm out of characters)